Search Results - "Cardoso, Cristina Ribeiro de Barros"

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  1. 1

    The Aryl Hydrocarbon Receptor (AHR) as a Potential Target for the Control of Intestinal Inflammation: Insights from an Immune and Bacteria Sensor Receptor by Pernomian, Larissa, Duarte-Silva, Murillo, de Barros Cardoso, Cristina Ribeiro

    Published in Clinical reviews in allergy & immunology (01-12-2020)
    “…The aryl hydrocarbon receptor (AHR) is widely expressed in immune and non-immune cells of the gut and its activation has been correlated to the outcome of…”
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    Journal Article
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    Microbiota Modulation of the Gut-Lung Axis in COVID-19 by de Oliveira, Gislane Lelis Vilela, Oliveira, Camilla Narjara Simão, Pinzan, Camila Figueiredo, de Salis, Larissa Vedovato Vilela, Cardoso, Cristina Ribeiro de Barros

    Published in Frontiers in immunology (24-02-2021)
    “…COVID-19 is an infectious disease caused by the Severe Acute Respiratory Syndrome Coronavirus-2 (SARS-CoV-2), and according to the World Health Organization…”
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    Toll‐like receptor 10 controls TLR2‐induced cytokine production in monocytes from patients with Parkinson's disease by Rocha Sobrinho, Hermínio Maurício, Gomes, Rodrigo, Silva, Delson José, Quixabeira, Valéria Bernadete Leite, Joosten, Leo A. B., Cardoso, Cristina, Ribeiro‐Dias, Fátima

    Published in Journal of neuroscience research (01-10-2021)
    “…Peripheral inflammation, particularly mediated by monocytes, can cause neuroinflammation in Parkinson's disease (PD). We investigated the mechanism of…”
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    CCR5 controls immune and metabolic functions during Toxoplasma gondii infection by Bonfá, Giuliano, Benevides, Luciana, Souza, Maria do Carmo, Fonseca, Denise Morais, Mineo, Tiago Wilson Patriarca, Rossi, Marcos Antônio, Silva, Neide Maria, Silva, João Santana, de Barros Cardoso, Cristina Ribeiro

    Published in PloS one (13-08-2014)
    “…CCR5, an important receptor related to cell recruitment and inflammation, is expressed during experimental Toxoplasma gondii infection. However, its role in…”
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    Inducible nitric oxide synthase controls experimental Strongyloides infection by Rodrigues, Rosângela Maria, Gonçalves, Ana Lúcia Ribeiro, Silva, Neide Maria, Cardoso, Cristina Ribeiro de Barros, Araújo, Natália Rodrigues, Coutinho, Loyane Bertagnolli, Alves, Ronaldo, Ueta, Marlene Tiduko, Costa‐Cruz, Julia Maria

    Published in Parasite immunology (01-10-2018)
    “…Summary Infection with Strongyloides sp. induces a host immune response, predominantly the Th2 type, that is able to eliminate the parasite. However, little is…”
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    New Horizons on Molecular Pharmacology Applied to Drug Discovery: When Resonance Overcomes Radioligand Binding by Pernomian, Larissa, Gomes, Mayara Santos, Moreira, Josimar Dornelas, da Silva, Carlos Henrique Tomich de Paula, Rosa, Joaquin Maria Campos, Cardoso, Cristina Ribeiro de Barros

    Published in Current radiopharmaceuticals (01-04-2017)
    “…One of the cornerstones of rational drug development is the measurement of molecular parameters derived from ligand-receptor interaction, which guides…”
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    Reappraisal of antibodies against Saccharomyces cerevisiae (ASCA) as persistent biomarkers in quiescent Crohn's disease by Duarte-Silva, Murillo, Afonso, Poliana Cristina, de Souza, Patrícia Reis, Peghini, Bethânea Crema, Rodrigues-Júnior, Virmondes, de Barros Cardoso, Cristina Ribeiro

    Published in Autoimmunity (Chur, Switzerland) (02-01-2019)
    “…A clear correlation exists between microbiota and the dysregulation of the immune response in Inflammatory Bowel Diseases (IBD), which comprise Crohn's disease…”
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    Dehydroepiandrosterone (DHEA) restrains intestinal inflammation by rendering leukocytes hyporesponsive and balancing colitogenic inflammatory responses by Alves, Vanessa Beatriz Freitas, Basso, Paulo José, Nardini, Viviani, Silva, Angélica, Chica, Javier Emílio Lazo, Cardoso, Cristina Ribeiro de Barros

    Published in Immunobiology (1979) (01-09-2016)
    “…Highlights • DHEA renders immune cells hyporesponsive in vitro and in vivo. • DHEA controls colitis by reducing leukocytes and inflammatory mediators in the…”
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    Journal Article