Search Results - "Capasso, Joseph M"

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  1. 1

    Structural remodeling and mechanical dysfunction of cardiac myocytes in heart failure by Gerdes, A.Martin, Capasso, Joseph M

    “…End stage heart failure due to ischemic (ICM) or dilated (DCM) cardiomyopathy is characterized by a dilated, relatively thin-walled ventricle. The hypothesis…”
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    Book Review Journal Article
  2. 2

    Cellular basis of ventricular remodeling after myocardial infarction by Anversa, Piero, Olivetti, Giorgio, Capasso, Joseph M.

    Published in The American journal of cardiology (18-11-1991)
    “…To determine whether acute left ventricular failure associated with myocardial infarction leads to architectural changes in the spared nonischemic portion of…”
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    Journal Article Conference Proceeding
  3. 3

    Regulation of Angiotensin II Receptors on Ventricular Myocytes After Myocardial Infarction in Rats by Meggs, Leonard G, Coupet, Joseph, Huang, Harer, Cheng, Wei, Li, Peng, Capasso, Joseph M, Homcy, Charles J, Anversa, Piero

    Published in Circulation research (01-06-1993)
    “…To determine the effects of acute myocardial infarction on the regulation of angiotensin II (Ang II) receptors and contractile performance of left and right…”
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    Journal Article
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    Upregulation of IGF1, IGF1-receptor, and late growth related genes in ventricular myocytes acutely after infarction in rats by Reiss, K, Meggs, L G, Li, P, Olivetti, G, Capasso, J M, Anversa, P

    Published in Journal of cellular physiology (01-01-1994)
    “…To determine the effects of acute myocardial infarction on the expression of insulin-like growth factor1 (IGF1) and insulin-like growth factor1 receptors…”
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    Journal Article
  6. 6

    Myocardial Mechanical, Biochemical, and Structural Alterations Induced by Chronic Ethanol Ingestion in Rats by Capasso, Joseph M, Li, Peng, Guideri, Giancarlo, Malhotra, Ashwani, Cortese, Rita, Anversa, Piero

    Published in Circulation research (01-08-1992)
    “…To determine the effects of moderate ethanol consumption on the mechanical, biochemical, and structural characteristics of the heart, myocardial mechanical…”
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    Journal Article
  7. 7

    Ventricular Loading Is Coupled With DNA Synthesis in Adult Cardiac Myocytes After Acute and Chronic Myocardial Infarction in Rats by Capasso, Joseph M, Bruno, Silvia, Cheng, Wei, Li, Peng, Rodgers, Robert, Darzynkiewicz, Zbigniew, Anversa, Piero

    Published in Circulation research (01-12-1992)
    “…To determine whether the overload associated with myocardial infarction and ventricular failure in rats is coupled with activation of DNA synthesis in the…”
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    Journal Article
  8. 8

    Efficacy of angiotensin-converting enzyme inhibition and AT1 receptor blockade on cardiac pump performance after myocardial infarction in rats by Capasso, J M, Li, P, Meggs, L G, Herman, M V, Anversa, P

    Published in Journal of cardiovascular pharmacology (01-04-1994)
    “…To determine whether cardiac unloading by inhibition of angiotensin I (AI) to AII conversion by captopril or blockade of the AII receptor (AT1) by losartan was…”
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    Journal Article
  9. 9

    Chronic Nonocclusive Coronary Artery Constriction Impairs Ventricular Function, Myocardial Structure, and Cardiac Contractile Protein Enzyme Activity in Rats by Capasso, Joseph M, Malhotra, Ashwani, Li, Peng, Zhang, Xun, Scheuer, James, Anversa, Piero

    Published in Circulation research (01-01-1992)
    “…To determine the effects of chronic nonocclusive coronary constriction on cardiac hemodynam-ics, structural integrity, and contractile protein enzyme activity,…”
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    Journal Article
  10. 10

    Myocyte loss and left ventricular failure characterise the long term effects of coronary artery narrowing or renal hypertension in rats by Li, P, Zhang, X, Capasso, J M, Meggs, L G, Sonnenblick, E H, Anversa, P

    Published in Cardiovascular research (01-06-1993)
    “…The aim was to determine the effects of chronic coronary artery narrowing and two kidney, one clip renal hypertension alone and in combination on ventricular…”
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    Journal Article
  11. 11

    Rapid Transition of Cardiac Myocytes from Hyperplasia to Hypertrophy During Postnatal Development by Li, Faqian, Wang, Xuejun, Capasso, Joseph M., Gerdes, A.Martin

    “…The switch from myocyte hyperplasia to hypertrophy occurs during the early postnatal period. The exact temporal sequence when cardiac myocytes cease dividing…”
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    Journal Article
  12. 12

    Effects of genetic hypertension and nutritional anaemia on ventricular remodelling and myocardial damage in rats by Olivetti, G, Quaini, F, Lagrasta, C, Ricci, R, Tosini, P, Capasso, J M, Anversa, P

    Published in Cardiovascular research (01-07-1993)
    “…In order to determine whether alterations in cardiac function and structure occur early in life in spontaneously hypertensive rats (SHR) and whether the…”
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    Journal Article
  13. 13

    Microvascular spasm as a cause of cardiomyopathies and the calcium-blocking agent verapamil as potential primary therapy by Sonnenblick, Edmund H., Fein, Frederick, Capasso, Joseph M., Factor, Stephen M.

    Published in The American journal of cardiology (01-01-1985)
    “…The origin of cardiomyopathies, a major cause of cardiac disability and death, has been largely unexplained. Pathologic features, common to all…”
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    Journal Article
  14. 14

    Size and shape of enzymatically isolated ventricular myocytes from rats and cardiomyopathic hamsters by Sorenson, A L, Tepper, D, Sonnenblick, E H, Robinson, T F, Capasso, J M

    Published in Cardiovascular research (01-12-1985)
    “…Rod-shaped and branched ventricular myocytes from rats and cardiomyopathic hamsters (strain 53.58) were isolated enzymatically, and their widths and lengths…”
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    Journal Article
  15. 15

    Cardiomyopathy of the Aging Human Heart: Myocyte Loss and Reactive Cellular Hypertrophy by Olivetti, Giorgio, Melissari, Massimo, Capasso, Joseph M, Anversa, Piero

    Published in Circulation research (01-06-1991)
    “…To determine the effects of aging on the human myocardium, 67 hearts were obtained from individuals who died from causes other than cardiovascular disease. The…”
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    Journal Article
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    Myocyte Cell Loss and Myocyte Cellular Hyperplasia in the Hypertrophied Aging Rat Heart by Anversa, Piero, Palackal, Thomas, Sonnenblick, Edmund H, Olivetti, Giorgio, Meggs, Leonard G, Capasso, Joseph M

    Published in Circulation research (01-10-1990)
    “…To determine the effects of age on the myocardium, the functional and structural characteristics of the heart were studied in rats at 4, 12, 20, and 29 months…”
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    Journal Article
  17. 17

    Side-to-Side Slippage of Myocytes Participates in Ventricular Wall Remodeling Acutely After Myocardial Infarction in Rats by Olivetti, Giorgio, Capasso, Joseph M, Sonnenblick, Edmund H, Anversa, Piero

    Published in Circulation research (01-07-1990)
    “…To determine whether acute left ventricular failure associated with myocardial infarction leads to architectural changes in the spared nonischemic portion of…”
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    Journal Article
  18. 18

    Effects of experimental phosphate deficiency on action potential characteristics and contractile performance of rat myocardium by Capasso, J M, Aronson, R S, Strobeck, J E, Sonnenblick, E H

    Published in Cardiovascular research (01-02-1982)
    “…Chronic hypophosphataemia is associated with reversible depression in myocardial performance of the intact heart. To define the basis for this, were studied…”
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    Journal Article
  19. 19

    Cellular Basis of Chronic Ventricular Remodeling After Myocardial Infarction in Rats by Olivetti, Giorgio, Capasso, Joseph M, Meggs, Leonard G, Sonnenblick, Edmund H, Anversa, Piero

    Published in Circulation research (01-03-1991)
    “…To determine whether the hypertrophic response of the surviving myocardium after infarction leads to normalization of ventricular hemodynamics and wall stress,…”
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    Journal Article
  20. 20

    Left Ventricular Failure Induced by Long-term Hypertension in Rats by Capasso, Joseph M, Palackal, Thomas, Olivetti, Giorgio, Anversa, Piero

    Published in Circulation research (01-05-1990)
    “…To determine whether the duration of hypertension is an essential component in the evolution of myocardial dysfunction, renal artery constriction was performed…”
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    Journal Article