Search Results - "Capasso, J. M."
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Cardiomyopathy of the Aging Human Heart: Myocyte Loss and Reactive Cellular Hypertrophy
Published in Circulation research (01-06-1991)“…To determine the effects of aging on the human myocardium, 67 hearts were obtained from individuals who died from causes other than cardiovascular disease. The…”
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2
The Expression of the γ Subunit of Na-K-ATPase is Regulated by Osmolality via C-Terminal Jun Kinase and Phosphatidylinositol 3-Kinase-Dependent Mechanisms
Published in Proceedings of the National Academy of Sciences - PNAS (06-11-2001)“…The α and β subunits of Na-K-ATPase are up-regulated by hypertonicity in inner-medullary collecting duct cells adapted to survive in hypertonic conditions. We…”
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3
Long-term adaptation of renal cells to hypertonicity: role of MAP kinases and Na-K-ATPase
Published in American journal of physiology. Renal physiology (01-05-2001)“…Renal cells in culture have low viability when exposed to hypertonicity. We developed cell lines of inner medullary collecting duct cells adapted to live at…”
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Myocyte Cell Loss and Myocyte Cellular Hyperplasia in the Hypertrophied Aging Rat Heart
Published in Circulation research (01-10-1990)“…To determine the effects of age on the myocardium, the functional and structural characteristics of the heart were studied in rats at 4, 12, 20, and 29 months…”
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Side-to-Side Slippage of Myocytes Participates in Ventricular Wall Remodeling Acutely After Myocardial Infarction in Rats
Published in Circulation research (01-07-1990)“…To determine whether acute left ventricular failure associated with myocardial infarction leads to architectural changes in the spared nonischemic portion of…”
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6
Cellular Basis of Chronic Ventricular Remodeling After Myocardial Infarction in Rats
Published in Circulation research (01-03-1991)“…To determine whether the hypertrophic response of the surviving myocardium after infarction leads to normalization of ventricular hemodynamics and wall stress,…”
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7
Different ways to die: cell death modes of the unicellular chlorophyte Dunaliella viridis exposed to various environmental stresses are mediated by the caspase-like activity DEVDase
Published in Journal of experimental botany (01-03-2009)“…Programmed cell death is necessary for homeostasis in multicellular organisms and it is also widely recognized to occur in unicellular organisms. However, the…”
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Regulation of Angiotensin II Receptors on Ventricular Myocytes After Myocardial Infarction in Rats
Published in Circulation research (01-06-1993)“…To determine the effects of acute myocardial infarction on the regulation of angiotensin II (Ang II) receptors and contractile performance of left and right…”
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Extensive digestion of Na+,K(+)-ATPase by specific and nonspecific proteases with preservation of cation occlusion sites
Published in The Journal of biological chemistry (15-01-1992)“…This paper extends our recent report that renal Na+,K(+)-ATPase is digested by trypsin in the absence of Ca2+ and presence of Rb+ ions to a stable 19-kDa…”
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10
ANG II receptors, c-myc, and c-jun in myocytes after myocardial infarction and ventricular failure
Published in The American journal of physiology (01-03-1993)“…To determine the relationship between reactive cardiac hypertrophy and the expression of angiotensin II (ANG II) receptors in surviving myocytes after…”
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11
Left Ventricular Failure Induced by Long-term Hypertension in Rats
Published in Circulation research (01-05-1990)“…To determine whether the duration of hypertension is an essential component in the evolution of myocardial dysfunction, renal artery constriction was performed…”
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12
Severe myocardial dysfunction induced by ventricular remodeling in aging rat hearts
Published in The American journal of physiology (01-10-1990)“…To determine if aging engenders alterations in the functional properties of the myocardium and ventricular remodeling, the hemodynamic performance and…”
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13
Loss of intermediate-sized coronary arteries and capillary proliferation after left ventricular failure in rats
Published in The American journal of physiology (01-05-1991)“…To determine the adaptation of coronary vasculature and microvasculature to cardiac failure, renal hypertension was produced in rats, and the animals were…”
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14
Myocyte Mitotic Division in the Aging Mammalian Rat Heart
Published in Circulation research (01-10-1991)“…To determine whether myocyte mitotic division occurs in the adult mammalian heart and whether this cellular process is affected by aging, we measured the…”
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15
Ischaemic myocardial injury and ventricular remodelling
Published in Cardiovascular research (01-02-1993)Get more information
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16
Cellular basis of ventricular remodeling after myocardial infarction
Published in The American journal of cardiology (18-11-1991)“…To determine whether acute left ventricular failure associated with myocardial infarction leads to architectural changes in the spared nonischemic portion of…”
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Journal Article Conference Proceeding -
17
Mechanical performance of spared myocytes after myocardial infarction in rats: effects of captopril treatment
Published in The American journal of physiology (01-09-1992)“…To determine the effects of myocardial infarction-induced left ventricular failure on the mechanical characteristics of the remaining viable myocytes, coronary…”
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18
Regression of cardiac hypertrophy after closing an aortocaval fistula in rats
Published in The American journal of physiology (01-06-1995)“…To determine whether the series addition of sarcomeres observed during eccentric hypertrophic growth is reversible upon removal of the initiating stimulus, an…”
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Cytosolic calcium transients in myocytes isolated from rats with ischemic heart failure
Published in The American journal of physiology (01-12-1993)“…Mechanical performance and cytosolic Ca2+ dynamics were characterized in myocytes isolated from left and right ventricles of rats with ischemic heart failure…”
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20
Alterations of coronary blood flow and reserve with aging in Fischer 344 rats
Published in The American journal of physiology (01-01-1989)“…To determine whether aging affects the coronary circulation, left and right ventricular coronary blood flow and vascular resistance at rest and after maximal…”
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