Search Results - "Canet, Luz M"

Polymorphisms at phase I-metabolizing enzyme and hormone receptor loci influence the response to anti-TNF therapy in rheumatoid arthritis patients by Canet, Luz M., Sánchez-Maldonado, Jose M., Cáliz, Rafael, Rodríguez-Ramos, Ana, Lupiañez, Carmen B., Canhao, Helena, Martínez-Bueno, Manuel, Escudero, Alejandro, Segura-Catena, Juana, Sorensen, Signe B, Hetland, Merete L, Soto-Pino, María José, Ferrer, Miguel A., García, Antonio, Glintborg, Bente, Filipescu, Ileana, Pérez-Pampin, Eva, González-Utrilla, Alfonso, Nevot, Miguel Ángel López, Conesa-Zamora, Pablo, Broeder, Alfons den, De Vita, Salvatore, Jacobsen, Sven Erik Hobe, Collantes-Estevez, Eduardo, Quartuccio, Luca, Canzian, Federico, Fonseca, João, Coenen, Marieke J. H., Andersen, Vibeke, Sainz, Juan

“…© Springer Nature Limited 2018 The aim of this case-control study was to evaluate whether 47 single-nucleotide polymorphisms (SNPs) in steroid hormone-related…”
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Genetic variants within the TNFRSF1B gene and susceptibility to rheumatoid arthritis and response to anti-TNF drugs: a multicenter study by Canet, Luz M, Filipescu, Ileana, Cáliz, Rafael, Lupiañez, Carmen B, Canhão, Helena, Escudero, Alejandro, Segura-Catena, Juana, Soto-Pino, María J, Ferrer, Miguel A, García, Antonio, Romani, Lurdes, Pérez-Pampin, Eva, González-Utrilla, Alfonso, López Nevot, Miguel A, Collantes, Eduardo, Fonseca, João E, Sainz, Juan

“…BACKGROUNDRecent research suggests that genetic variants in the tumor necrosis factor receptor 2 (TNFRSF1B) gene may have an impact on susceptibility to…”
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Genetic variants within immune-modulating genes influence the risk of developing rheumatoid arthritis and anti-TNF drug response: a two-stage case–control study by Canet, Luz M, Cáliz, Rafael, Lupiañez, Carmen B, Canhão, Helena, Martinez, Manuel, Escudero, Alejandro, Filipescu, Ileana, Segura-Catena, Juana, Soto-Pino, María J, Ferrer, Miguel A, García, Antonio, Romani, Lurdes, Pérez-Pampin, Eva, González-Utrilla, Alfonso, López Nevot, Miguel Ángel, Collantes, Eduardo, Fonseca, João E, Sainz, Juan

“…BACKGROUNDRheumatoid arthritis (RA) is a chronic autoimmune disease that arises as a result of the interaction between genetic and environmental factors. A…”
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Correction: Polymorphisms at phase I-metabolizing enzyme and hormone receptor loci influence the response to anti-TNF therapy in rheumatoid arthritis patients by Canet, Luz M., Sánchez-Maldonado, Jose M., Cáliz, Rafael, Rodríguez-Ramos, Ana, Lupiañez, Carmen B., Canhão, Helena, Martínez-Bueno, Manuel, Escudero, Alejandro, Segura-Catena, Juana, Sorensen, Signe B., Hetland, Merete L., Soto-Pino, María José, Ferrer, Miguel A., García, Antonio, Glintborg, Bente, Filipescu, Ileana, Pérez-Pampin, Eva, González-Utrilla, Alfonso, Nevot, Miguel Ángel López, Conesa-Zamora, Pablo, den Broeder, Alfons, De Vita, Salvatore, Jacobsen, Sven Erik Hobe, Collantes-Estevez, Eduardo, Quartuccio, Luca, Canzian, Federico, Fonseca, João E., Coenen, Marieke J. H., Andersen, Vibeke, Sainz, Juan

“…The original version of this Article contained an error in the spelling of the author Ana Rodríguez-Ramos, which was incorrectly given as Ana Rodríguez Ramos…”
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Correction: Polymorphisms at phase I-metabolizing enzyme andhormone receptor loci influence the response to anti-TNF therapy in rheumatoid arthritispatients by Canet Luz M, Sánchez-Maldonado, Jose M, Cáliz Rafael, Rodríguez-Ramos, Ana, Lupiañez, Carmen B, Canhão Helena, Martínez-Bueno, Manuel, Escudero, Alejandro, Segura-Catena Juana, Sorensen, Signe B, Hetland, Merete L, Soto-Pino, María José, Ferrer, Miguel A, García, Antonio, Glintborg Bente, Filipescu Ileana, Pérez-Pampin, Eva, González-Utrilla, Alfonso, Nevot Miguel Ángel López, Conesa-Zamora, Pablo, den Broeder Alfons, De Vita Salvatore, Jacobsen Sven Erik Hobe, Collantes-Estevez, Eduardo, Quartuccio Luca, Canzian Federico, Fonseca, João E, Coenen Marieke J H, Andersen, Vibeke, Sainz, Juan

“…The original version of this Article contained an error in the spelling of the author Ana Rodríguez-Ramos, which was incorrectly given as Ana Rodríguez Ramos…”
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Common Genetic Polymorphisms within NFκB-Related Genes and the Risk of Developing Invasive Aspergillosis by Lupiañez, Carmen B, Villaescusa, María T, Carvalho, Agostinho, Springer, Jan, Lackner, Michaela, Sánchez-Maldonado, José M, Canet, Luz M, Cunha, Cristina, Segura-Catena, Juana, Alcazar-Fuoli, Laura, Solano, Carlos, Fianchi, Luana, Pagano, Livio, Potenza, Leonardo, Aguado, José M, Luppi, Mario, Cuenca-Estrella, Manuel, Lass-Flörl, Cornelia, Einsele, Hermann, Vázquez, Lourdes, Ríos-Tamayo, Rafael, Loeffler, Jurgen, Jurado, Manuel, Sainz, Juan

“…Invasive Aspergillosis (IA) is an opportunistic infection caused by Aspergillus, a ubiquitously present airborne pathogenic mold. A growing number of studies…”
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Genetic variants within immune-modulating genes influence the risk of developing rheumatoid arthritis and anti-TNF drug response by Canet, Luz M., Cáliz, Rafael, Lupiañez, Carmen B., Canhao, Helena, Martinez, Manuel, Escudero, Alejandro, Filipescu, Ileana, Segura-Catena, Juana, Soto-Pino, María J., Ferrer, Miguel A., García, Antonio, Romani, Lurdes, Pérez-Pampin, Eva, González-Utrilla, Alfonso, López Nevot, Miguel Ángel, Collantes, Eduardo, Fonseca, João, Sainz, Juan

“…Copyright © 2015 Wolters Kluwer Health, Inc. All rights reserved. Background: Rheumatoid arthritis (RA) is a chronic autoimmune disease that arises as a result…”
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