Search Results - "Caillaux, G"

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  1. 1

    P88 – 2855: Deficient alpha-dystroglycan presenting with rhabdomyolysis by Kuster, A, Caillaux, G, Leturcq, F, Romero, N

    Published in European journal of paediatric neurology (01-05-2015)
    “…Objective To describe a case of deficient alpha-dystroglycan revealed by acute rhabdomyolysis. Rhabdomyolysis frequently occurs in metabolic myopathies induced…”
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    Journal Article
  2. 2

    Hospital initiation of a vaccinal schedule improves the long-term vaccinal coverage of ex-preterm children by Denizot, S, Fleury, J, Caillaux, G, Rouger, V, Rozé, J.-C, Guen, C. Gras-Le

    Published in Vaccine (10-01-2011)
    “…Abstract Preterm infants are experiencing delays in receiving routine schedule vaccines. We evaluated up-to-date immunisation status of 602 preterm infants at…”
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    Journal Article
  3. 3

    Neonatal surgical antibioprophylaxis: which target? which propositions? by Gras-Le Guen, C, Dumont, R, Pichenot, V, Caillaux, G, Caillon, J, Leclair, M-D, de La Cochetière, M-F, Launay, E

    “…Neonatal surgical antibioprophylaxis must take into account the specificities of this life period, as immunological immaturity, microbiotal implantation and…”
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    Journal Article
  4. 4

    Expanding the spectrum of congenital myopathy linked to recessive mutations in SCN4A by Mercier, Sandra, Lornage, Xavière, Malfatti, Edoardo, Marcorelles, Pascale, Letournel, Franck, Boscher, Cécile, Caillaux, Gaëlle, Magot, Armelle, Böhm, Johann, Boland, Anne, Deleuze, Jean-François, Romero, Norma, Péréon, Yann, Laporte, Jocelyn

    Published in Neurology (24-01-2017)
    “…Congenital myopathies are phenotypically and genetically heterogeneous.1 While SCN4A mutations were previously described in hypokalemic or hyperkalemic…”
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    Antibioprophylaxie en chirurgie néonatale : quelle est la cible ? quelles propositions ? by Gras-Le Guen, C., Dumont, R., Pichenot, V., Caillaux, G., Caillon, J., Leclair, M.-D., de La Cochetière, M.-F., Launay, E.

    “…L’antibioprophylaxie en chirurgie néonatale doit tenir compte des spécificités de cette période de la vie, portant en particulier l’immaturité immunitaire et…”
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    SFP-P184 – Métabolisme – Mode de révélation atypique d’un déficit du cycle de l’urée avec hyperlactacidémie majeure by Baruteau, J., Valayannopoulos, V., De Lonlay, P., Caillaux, G., Liet, J.-M., Dejode, J.-M., Roze, J.-C., Vianey-Saban, C., Haberle, J., Kuster, A.

    “…Le cycle de l’urée permet l’élimination de l’ammoniaque issu du catabolisme protéique par sa transformation en urée. Le déficit d’une de ses enzymes se révèle…”
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