Search Results - "Cabitta, Livia"

Sphingosine 1-Phosphate Receptors and Metabolic Enzymes as Druggable Targets for Brain Diseases by Grassi, Sara, Mauri, Laura, Prioni, Simona, Cabitta, Livia, Sonnino, Sandro, Prinetti, Alessandro, Giussani, Paola

“…The central nervous system is characterized by a high content of sphingolipids and by a high diversity in terms of different structures. Stage- and…”
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The Role of 3-O-Sulfogalactosylceramide, Sulfatide, in the Lateral Organization of Myelin Membrane by Grassi, Sara, Prioni, Simona, Cabitta, Livia, Aureli, Massimo, Sonnino, Sandro, Prinetti, Alessandro

“…Sulfatide (3- O -sulfogalactosylceramide, SM4s) was isolated by Thudichum from the human brain in 1884. Together with galactosylceramide, its direct metabolic…”
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Identification of the Lipid Antigens Recognized by rHIgM22, a Remyelination-Promoting Antibody by Grassi, Sara, Cabitta, Livia, Prioni, Simona, Mauri, Laura, Ciampa, Maria Grazia, Yokoyama, Noriko, Iwabuchi, Kazuhisa, Zorina, Yana, Prinetti, Alessandro

“…Failure of the immune system to discriminate myelin components from foreign antigens plays a critical role in the pathophysiology of multiple sclerosis. In…”
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Human Remyelination Promoting Antibody Stimulates Astrocytes Proliferation Through Modulation of the Sphingolipid Rheostat in Primary Rat Mixed Glial Cultures by Grassi, Sara, Giussani, Paola, Prioni, Simona, Button, Donald, Cao, Jing, Hakimi, Irina, Sarmiere, Patrick, Srinivas, Maya, Cabitta, Livia, Sonnino, Sandro, Prinetti, Alessandro

“…Remyelination promoting human IgMs effectively increase the number of myelinated axons in animal models of multiple sclerosis. Hence, they ultimately stimulate…”
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A pathogenic HEXA missense variant in wild boars with Tay-Sachs disease by Bertani, Valeria, Prioni, Simona, Di Lecce, Rosanna, Gazza, Ferdinando, Ragionieri, Luisa, Merialdi, Giuseppe, Bonilauri, Paolo, Jagannathan, Vidhya, Grassi, Sara, Cabitta, Livia, Paoli, Antonella, Morrone, Amelia, Sonnino, Sandro, Drögemüller, Cord, Cantoni, Anna Maria

“…Gangliosidoses are inherited lysosomal storage disorders caused by reduced or absent activity of either a lysosomal enzyme involved in ganglioside catabolism,…”
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Identification of the antigen recognized by rHIgM22, a remyelination-promoting human monoclonal antibody by Grassi, Sara, Prioni, Simona, Zorina, Yana, Cabitta, Livia, Sonnino, Sandro, Prinetti, Alessandro

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