Search Results - "CALO, L. A"
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The importance of chronic magnesium deficiency in human disease and the Gitelman's syndrome paradox
Published in QJM : An International Journal of Medicine (01-06-2019)Get full text
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Mechanistic approach to the pathophysiology of target organ damage in hypertension from studies in a human model with characteristics opposite to hypertension: Bartter’s and Gitelman’s syndromes
Published in Journal of endocrinological investigation (01-07-2015)“…Introduction Extensive studies using Bartter’s/Gitelman’s syndrome patients have provided insights into the angiotensin II (Ang II) signaling pathways involved…”
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Vascular tone control in humans: Insights from studies in Bartter's/Gitelman's syndromes
Published in Kidney international (01-03-2006)“…Studies in patients with Bartter's and Gitelman's syndromes performed in the last 10 years have provided important insights into the mechanistic details of…”
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Aborted sudden cardiac death in two patients with Bartter's/Gitelman's syndromes
Published in Clinical nephrology (01-03-2007)“…Sudden cardiac death (SCD) occurs in patients with Bartter/Gitelman syndromes. Hypokalemia and QTc prolongation are suggested mechanisms. SCD, however, has…”
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Myocardial perfusion defects in Bartter and Gitelman syndromes
Published in European journal of clinical investigation (01-12-2008)“…ABSTRACT Background Normotensive hypokalaemic tubulopathies (Bartter and Gitelman syndromes (BS/GS)) are genetic diseases that are considered benign. However,…”
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Rho kinase, oxidative stress, ACE2/Ang 1-7 and lung fibrosis
Published in Minerva medica (01-06-2015)Get more information
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Phosphorus in excess: a 'nutrient pollution' problem in the blood-broken hearts like broken lakes (eutrophication)?
Published in European journal of clinical nutrition (01-12-2009)Get full text
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Linking inflammation and hypertension in humans: studies in Bartter's Gitelman's syndrome patients
Published in Journal of human hypertension (01-03-2008)Get full text
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Severe diarrhea and the McKittrick and Wheelock syndrome
Published in QJM : An International Journal of Medicine (29-04-2023)Get full text
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PHOS-FAKE or PHOS-FATAL - does intake of phosphate from foods lead to elevated plasma phosphate and its associated cardiovascular risks?
Published in Clinical nephrology (01-07-2010)Get more information
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A new approach to the evaluation of hyperphosphatemia in chronic kidney disease
Published in Clinical nephrology (01-10-2007)“…Hyperphosphoremia, main contributor to cardiovascular calcifications, has a major impact on the morbidity and mortality of chronic renal failure (CRF)…”
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Echocardiography might not be able to detect left ventricular hypertrophy in all dialysis patients
Published in Minerva medica (01-04-2012)Get more information
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Erythropoietin, heme oxygenase-1 and vascular tissue protective effects
Published in Clinical nephrology (01-03-2009)Get more information
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Aldosterone and thrombosis formation: implications for ischemic and atherosclerotic heart disease
Published in Journal of endocrinological investigation (01-07-2006)Get full text
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Hemodiafiltration and oxidative stress in dialysis patients
Published in Blood purification (01-01-2009)Get more information
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Proinflammatory/profibrotic effects of aldosterone in Gitelman’s syndrome, a human model opposite to hypertension
Published in Journal of endocrinological investigation (01-05-2019)“…Purpose Aldosterone proinflammatory/profibrotic effects are mediated by the induction of mononuclear leucocytes (MNL) to express oxidative stress…”
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Validation of an easy questionnaire on the assessment of salt habit: the MINISAL-SIIA Study Program
Published in European journal of clinical nutrition (01-05-2019)“…Background/objectives The aim of the present study was to validate a short questionnaire on habitual dietary salt intake, to quickly and easily identify…”
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Gαq/p63RhoGEF interaction in RhoA/Rho kinase signaling: investigation in Gitelman’s syndrome and implications with hypertension
Published in Journal of endocrinological investigation (01-03-2018)“…Purpose Gitelman’s syndrome (GS) presents normo-hypotension and absence of cardiovascular–renal remodeling despite high angiotensin II (Ang II), activation of…”
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