Search Results - "Brotto, Marco A. P."

Functional and biochemical modifications in skeletal muscles from malarial mice by Brotto, Marco A. P., Marrelli, Mauro T., Brotto, Leticia S., Jacobs‐Lorena, Marcelo, Nosek, Thomas M.

“…Although it is well established that patients suffering from malaria experience skeletal muscle problems (contracture, aches, fatigue, weakness), detailed…”
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Increased susceptibility to fatigue of slow- and fast-twitch muscles from mice lacking the MG29 gene by NAGARAJ, RAMAKRISHNAN Y, NOSEK, CHRISTOPHER M, BROTTO, MARCO A. P, NISHI, MIYUKI, TAKESHIMA, HIROSHI, NOSEK, THOMAS M, MA, JIANJIE

“…1 Department of Physiology and Biophysics, Case Western Reserve University, School of Medicine, Cleveland, Ohio 44106-4963 2 Division of Cell Biology,…”
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Functional properties of skeletal muscle from transgenic animals with upregulated heat shock protein 70 by NOSEK, THOMAS M, BROTTO, MARCO A. P, ESSIG, DAVID A, MESTRIL, RUBEN, CONOVER, RICHARD C, DILLMANN, WOLFGANG H, KOLBECK, RALPH C

“…1 Department of Physiology and Biophysics, Case Western Reserve University School of Medicine, Cleveland, Ohio 44106-4963 2 Department of Biology, Geneva…”
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Effects of PI(3,5)P2 on the Skeletal Muscle Ryanodine Receptor by Brotto, Marco A. P., Sherman, Joseph A., Nosek, Thomas M, Qu, Cheng Kui, Valdivia, Héctor H

“…Abstract only An increase of intracellular PI(3,5)P2 in MIP KO mice is associated with a loss of calcium (Ca) from the SR. Is this loss of Ca due to Ca leakage…”
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Defective maintenance of intracellular Ca2+ homeostasis is linked to increased muscle fatigability in the MG29 null mice by Brotto, Marco A P, Nagaraj, Ramakrishnan Y, Brotto, Leticia S, Takeshima, Hiroshi, Ma, Jian Jie, Nosek, Thomas M

“…Mitsugumin 29 (MG29) is a transmembrane protein that is normally found in the triad junction of skeletal muscle. Our previous studies have shown that targeted…”
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Excitation-contraction coupling in the day 15 embryonic chick heart with persistent truncus arteriosus by CREAZZO, T. L, BROTTO, M. A. P, BURCH, J

“…Ca2+ transients were examined in embryonic chick hearts with an experimentally induced cardiac neural crest-related outflow tract defect known as persistent…”
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