Search Results - "Brishti, Masuma Akter"

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  1. 1

    Hypoxia induces purinergic receptor signaling to disrupt endothelial barrier function by Raghavan, Somasundaram, Brishti, Masuma Akter, Collier, Daniel Mohr, Leo, M Dennis

    Published in Frontiers in physiology (21-11-2022)
    “…Blood-brain-barrier permeability is regulated by endothelial junctional proteins and is vital in limiting access to and from the blood to the CNS. When…”
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    Journal Article
  2. 2

    Diabetic Endothelial Cell Glycogen Synthase Kinase 3β Activation Induces VCAM1 Ectodomain Shedding by Brishti, Masuma Akter, Raghavan, Somasundaram, Lamar, Kennedy, Singh, Udai P, Collier, Daniel M, Leo, M. Dennis

    “…Soluble cell adhesion molecules (sCAMs) are secreted ectodomain fragments of surface adhesion molecules, ICAM1 and VCAM1. sCAMs have diverse immune functions…”
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    Journal Article
  3. 3

    Rab GTPases as Modulators of Vascular Function by Raghavan, Somasundaram, Brishti, Masuma Akter, Leo, M. Dennis

    Published in Cells (Basel, Switzerland) (29-09-2022)
    “…Rab GTPases, the largest family of small GTPases, are ubiquitously expressed proteins that control various aspects of cellular function, from cell survival to…”
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    Journal Article
  4. 4

    Abstract 4133170: Insulin resistance triggers histamine release from endothelial cells, which disrupts endothelial barrier function and causes vasoconstriction by Brishti, Masuma Akter, Leo, M

    Published in Circulation (New York, N.Y.) (12-11-2024)
    “…Abstract only Diabetic vascular dysfunction is a serious health concern, affecting ~38 million people in the USA, according to the 2020 CDC-National Diabetes…”
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    Journal Article
  5. 5

    Extracellular glucose and dysfunctional insulin receptor signaling independently upregulate arterial smooth muscle TMEM16A expression by Raghavan, Somasundaram, Brishti, Masuma Akter, Bernardelli, Angelica, Mata-Daboin, Alejandro, Jaggar, Jonathan H, Leo, M Dennis

    “…Diabetes alters the function of ion channels responsible for regulating arterial smooth muscle membrane potential, resulting in vasoconstriction. Our prior…”
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    Journal Article