Search Results - "Bréchard, S"

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  1. 1

    Calcium signaling and regulation of neutrophil functions: Still a long way to go by Hann, J., Bueb, J.‐L., Tolle, F., Bréchard, S.

    Published in Journal of leukocyte biology (01-02-2020)
    “…Neutrophils are the most abundant leukocytes in blood and disruption in their functions often results in an increased risk of serious infections and…”
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    Journal Article
  2. 2

    Regulation of neutrophil pro-inflammatory functions sheds new light on the pathogenesis of rheumatoid arthritis by Jung, N., Bueb, J.-L., Tolle, F., Bréchard, S.

    Published in Biochemical pharmacology (01-07-2019)
    “…[Display omitted] For more than two centuries now, rheumatoid arthritis (RA) is under investigation intending to discover successful treatment. Despite decades…”
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    Journal Article
  3. 3

    Store-operated Ca2+ channels formed by TRPC1, TRPC6 and Orai1 and non-store-operated channels formed by TRPC3 are involved in the regulation of NADPH oxidase in HL-60 granulocytes by Bréchard, S, Melchior, C, Plançon, S, Schenten, V, Tschirhart, E.J

    Published in Cell calcium (Edinburgh) (01-11-2008)
    “…Summary Ca2+ influx has been shown to be essential for NADPH oxidase activity which is involved in the inflammatory process. Ca2+ conditions underlying the…”
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    Journal Article
  4. 4

    STIM1 but not STIM2 is an essential regulator of Ca2+ influx-mediated NADPH oxidase activity in neutrophil-like HL-60 cells by Bréchard, S., Plançon, S., Melchior, C., Tschirhart, E.J.

    Published in Biochemical pharmacology (01-09-2009)
    “…STIM1, not STIM2, regulates Ca2+ influx-mediated NADPH oxidase activity in neutrophil-like HL-60 cells. Extracellular Ca2+ entry, primarily mediated through…”
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    Journal Article
  5. 5

    Interleukin-8 primes oxidative burst in neutrophil-like HL-60 through changes in cytosolic calcium by Bréchard, S., Bueb, J.-L., Tschirhart, E.J.

    Published in Cell calcium (Edinburgh) (01-06-2005)
    “…In response to a variety of stimuli, neutrophils release large amount of reactive oxygen species (ROS) generated by NADPH oxidase. This process known as the…”
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    Journal Article
  6. 6

    Modulation by cADPr of Ca2+ mobilization and oxidative response in dimethylsulfoxide- or retinoic acid-differentiated HL-60 cells by Bréchard, S, Brunello, A, Bueb, J-L, Tschirhart, E J

    Published in Biochimica et biophysica acta (01-01-2006)
    “…In human phagocytic cells, reactive oxygen species (ROS) generation in response to N-formyl-L-Methionyl-L-Leucyl-L-Phenylalanine (fMLF) is largely dependent on…”
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    Journal Article
  7. 7

    Correlation between tyrosine phosphorylation intensity of a 107 kDa protein band and A23187-induced acrosome reaction in human spermatozoa by Picherit-Marchenay, C., Bréchard, S., Boucher, D., Grizard, G.

    Published in Andrologia (01-12-2004)
    “…This study, performed using semen samples from 10 men, investigated the relationship between sperm protein tyrosine phosphorylation and acrosomal status in…”
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  10. 10

    Store-operated Ca 2+ channels formed by TRPC1, TRPC6 and Orai1 and non-store-operated channels formed by TRPC3 are involved in the regulation of NADPH oxidase in HL-60 granulocytes by Bréchard, S., Melchior, C., Plançon, S., Schenten, V., Tschirhart, E.J.

    Published in Cell calcium (Edinburgh) (2008)
    “…Ca 2+ influx has been shown to be essential for NADPH oxidase activity which is involved in the inflammatory process. Ca 2+ conditions underlying the oxidative…”
    Get full text
    Journal Article
  11. 11

    Modulation by cADPr of Ca 2+ mobilization and oxidative response in dimethylsulfoxide- or retinoic acid-differentiated HL-60 cells by Bréchard, S., Brunello, A., Bueb, J.-L., Tschirhart, E.J.

    “…In human phagocytic cells, reactive oxygen species (ROS) generation in response to N-formyl- l-Methionyl- l-Leucyl- l-Phenylalanine (fMLF) is largely dependent…”
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    Journal Article
  12. 12

    Regulation of superoxide production in neutrophils: role of calcium influx by Bréchard, Sabrina, Tschirhart, Eric J.

    Published in Journal of leukocyte biology (01-11-2008)
    “…Upon stimulation, activation of NADPH oxidase complexes in neutrophils produces a burst of superoxide anions contributing to oxidative stress and the…”
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    Journal Article
  13. 13

    OAG induces an additional PKC-, PI3K-, and Rac2-mediated signaling pathway up-regulating NOX2 activity, independently of Ca super(2+) entry by Brechard, S, Salsmann, A, Tschirhart, E J

    Published in Journal of leukocyte biology (01-04-2009)
    “…The requirement of calcium ion (Ca super(2+)) entry for neutrophil NADPH oxidase (NOX2) regulation is clearly established. However, its role in the signaling…”
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    Journal Article
  14. 14

    STIM1 but not STIM2 is an essential regulator of Ca super(2+) influx-mediated NADPH oxidase activity in neutrophil-like HL-60 cells by Brechard, S, Plancon, S, Melchior, C, Tschirhart, E J

    Published in Biochemical pharmacology (01-09-2009)
    “…Extracellular Ca super(2+) entry, primarily mediated through store-operated Ca super(2+) entry (SOCE), is known to be a critical event for NADPH oxidase (NOX2)…”
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    Journal Article
  15. 15

    DNA analysis as a tool to confirm the diagnosis of asymptomatic hereditary neuropathy with liability to pressure palsies (HNPP) with further evidence for the occurrence of de novo mutations by Gonnaud, P M, Sturtz, F, Fourbil, Y, Bonnebouche, C, Tranchant, C, Warter, J M, Chazot, G, Bady, B, Vial, C, Brechard, A S

    Published in Acta neurologica Scandinavica (01-10-1995)
    “…We performed DNA analysis in four families with hereditary neuropathy with liability to pressure palsy (HNPP). An interstitial deletion of the 17 p11.2 region…”
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    Journal Article
  16. 16

    Store-operated Ca super(2) super(+) channels formed by TRPC1, TRPC6 and Orai1 and non-store-operated channels formed by TRPC3 are involved in the regulation of NADPH oxidase in HL-60 granulocytes by Brechard, S, Melchior, C, Plancon, S, Schenten, V, Tschirhart, E J

    Published in Cell calcium (Edinburgh) (01-11-2008)
    “…Ca super(2) super(+) influx has been shown to be essential for NADPH oxidase activity which is involved in the inflammatory process. Ca super(2) super(+)…”
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    Journal Article
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