Search Results - "Bertocchi, A.P.F."

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  1. 1

    The role of heme oxygenase 1 in rapamycin-induced renal dysfunction after ischemia and reperfusion injury by Gonçalves, G.M., Cenedeze, M.A., Feitoza, C.Q., Wang, P.M.H., Bertocchi, A.P.F., Damião, M.J., Pinheiro, H.S., Antunes Teixeira, V.P., dos Reis, M.A., Pacheco-Silva, A., Câmara, N.O.S.

    Published in Kidney international (01-11-2006)
    “…Ischemia and reperfusion injury (IRI) is the main etiology of acute renal failure in native and transplanted kidneys. In the transplantation field,…”
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    Journal Article
  2. 2

    The Role of Toll-Like Receptor 4 in Cisplatin-Induced Renal Injury by Cenedeze, M.A, Gonçalves, G.M, Feitoza, C.Q, Wang, P.M.H, Damião, M.J, Bertocchi, A.P.F, Pacheco-Silva, A, Câmara, N.O.S

    Published in Transplantation proceedings (01-03-2007)
    “…Abstract Background Toll-like receptors recognize pattern-associated molecules found in pathogens as well as in endogen cells and in matrix degradation…”
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    Journal Article
  3. 3

    The Effects of Rapamycin in the Progression of Renal Fibrosis by Damião, M.J, Bertocchi, A.P.F, Monteiro, R.M.M, Gonçalves, G.M, Cenedeze, M.A, Feitoza, C.Q, Marques, G.D, Giannocco, G, Mazzali, M, Teixeira, V.P.A, dos Reis, M.A, Pacheco-Silva, A, Câmara, N.O.S

    Published in Transplantation proceedings (01-03-2007)
    “…Abstract Renal fibrosis is a hallmark of end-stage renal diseases and of chronic allograft nephropathy (CAN). Rapamycin, besides its action through blockade of…”
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    Journal Article Conference Proceeding
  4. 4

    A Role for HO-1 in Renal Function Impairment in Animals Subjected to Ischemic and Reperfusion Injury and Treated With Immunosuppressive Drugs by Feitoza, C.Q, Gonçalves, G.M, Bertocchi, A.P.F, Wang, P.W, Damião, M.J, Cenedeze, M.A, Teixeira, V.P.A, dos Reis, M.A, Pacheco-Silva, A, Câmara, N.O.S

    Published in Transplantation proceedings (01-03-2007)
    “…Abstract Introduction Ischemia/reperfusion injury (IRI) represents the single major antigen-independent factor implicated in pathogenesis of chronic graft…”
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    Journal Article Conference Proceeding