Search Results - "Berland, Robert"
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Listeria monocytogenes Infection Induces Prosurvival Metabolic Signaling in Macrophages
Published in Infection and Immunity (01-04-2011)“…Host cells use metabolic signaling through the LXRα nuclear receptor to defend against Listeria monocytogenes infection. 25-Hydroxycholesterol is a natural…”
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The sst1 resistance locus regulates evasion of type I interferon signaling by Chlamydia pneumoniae as a disease tolerance mechanism
Published in PLoS pathogens (01-08-2013)“…The sst1, "supersusceptibility to tuberculosis," locus has previously been shown to be a genetic determinant of host resistance to infection with the…”
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Normal B-1a Cell Development Requires B Cell-Intrinsic NFATc1 Activity
Published in Proceedings of the National Academy of Sciences - PNAS (11-11-2003)“…B-1a cells, an anatomically, phenotypically, and functionally distinct subset of B cells that produce the bulk of natural serum IgM and much of gut-associated…”
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The integrated stress response mediates necrosis in murine Mycobacterium tuberculosis granulomas
Published in The Journal of clinical investigation (01-02-2021)“…The mechanism by which only some individuals infected with Mycobacterium tuberculosis develop necrotic granulomas with progressive disease while others form…”
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Microbial Lipid A Remodeling Controls Cross-Presentation Efficiency and CD8 T Cell Priming by Modulating Dendritic Cell Function
Published in Infection and immunity (19-01-2021)“…The majority of Gram-negative bacteria elicit a potent immune response via recognition of lipid A expressed on the outer bacterial membrane by the host immune…”
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ORIGINS AND FUNCTIONS OF B-1 CELLS WITH NOTES ON THE ROLE OF CD5
Published in Annual review of immunology (01-01-2002)“…Whether B-1a (CD5+) cells are a distinct lineage derived from committed fetal/neonatal precursors or arise from follicular B-2 cells in response to BCR…”
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Toll-like Receptor 7-Dependent Loss of B Cell Tolerance in Pathogenic Autoantibody Knockin Mice
Published in Immunity (Cambridge, Mass.) (01-09-2006)“…Systemic lupus erythematosus (SLE) is characterized by the production of autoantibodies that are frequently directed against nucleic acid-associated antigens…”
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A conserved enhancer element differentially regulates developmental expression of CD5 in B and T cells
Published in The Journal of immunology (1950) (15-12-2010)“…We previously identified an enhancer element upstream of the mouse cd5 gene that was required in reporter assays for the induction of cd5 promoter activity by…”
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The sst1 Resistance Locus Regulates Evasion of Type I Interferon Signaling by Chlamydia pneumoniae as a Disease Tolerance Mechanism: e1003569
Published in PLoS pathogens (01-08-2013)“…The sst1, "supersusceptibility to tuberculosis," locus has previously been shown to be a genetic determinant of host resistance to infection with the…”
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10
Cutting Edge Commentary: Origins of B-1 Cells
Published in The Journal of immunology (1950) (15-02-2001)“…The origin of B-1a cells, a minority population of B cells that express CD5, are abundant in coelomic cavities, and often produce autoantibodies, has been the…”
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Vav-2 controls NFAT-dependent transcription in B- but not T-lymphocytes
Published in The EMBO journal (15-11-2000)“…We show here that Vav-2 is tyrosine phosphorylated following antigen receptor engagement in both B- and T-cells, but potentiates nuclear factor of activated T…”
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An NFAT-Dependent Enhancer Is Necessary for Anti-IgM-Mediated Induction of Murine CD5 Expression in Primary Splenic B Cells
Published in The Journal of immunology (1950) (01-07-1998)“…CD5 is a 67-kDa membrane glycoprotein the expression of which in murine splenic B cells is induced by surface IgM cross-linking. To analyze this induction, we…”
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A model for autoantigen induction of natural antibody producing B-1a cells
Published in Current topics in microbiology and immunology (2000)Get more information
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14
Vav-2 controls NFAT-dependent transcription inB- but not T-lymphocytes
Published in The EMBO journal (15-11-2000)“…We show here that Vav‐2 is tyrosine phosphorylated following antigen receptor engagement in both B‐ and T‐cells, but potentiates nuclear factor of activated T…”
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15
Serologic Diagnosis of Lyme Disease using Recombinant Outer Surface Proteins A and B and Flagellin
Published in The Journal of infectious diseases (01-06-1992)“…Recombinant (r) outer surface proteins (Osp) A and B, flagellin, and an immunogenic region of flagellin (41-G) from Borrelia burgdorferi were evaluated using…”
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Serologic response to the Borrelia burgdorferi flagellin demonstrates an epitope common to a neuroblastoma cell line
Published in Proceedings of the National Academy of Sciences - PNAS (01-01-1993)“…Antibodies in sera of 7 patients with neurologic manifestations of Lyme borreliosis and a monoclonal antibody (mAb H9724) to the flagellin of Borrelia…”
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Differential regulation of accessory mitogenic signaling receptors by the T cell antigen receptor
Published in Molecular immunology (01-04-1995)“…In addition to the antigen receptor, resting T cells express a number of receptors that can be stimulated to generate proliferative signals. These “accessory”…”
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Serologic Diagnosis of Lyme Disease using Recombinant Outer Surface Proteins A and Band Flagellin
Published in The Journal of infectious diseases (01-06-1992)“…Recombinant (r) outer surface proteins (Osp) A and B, flagellin, and an immunogenicregion of flagellin (41-G) from Borrelia burgdorferi were evaluated using…”
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The rat albumin gene promoter is appropriately regulated in transient but not in stable transfections
Published in Nucleic acids research (23-12-1988)“…The tissue-specific expression of the liver-specific rat albumin gene promoter was examined after transfer to various hepatic and non-hepatic cell lines. A 402…”
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Reinitiation of Translation from the Triplet next to the Amber Termination Codon in the Absence of Ribosome-Releasing Factor
Published in Proceedings of the National Academy of Sciences - PNAS (01-10-1981)“…Ribosome releasing factor (RR factor) releases ribosomes from mRNA at the termination codon in Escherichia coli. In the absence of this factor, polypeptides…”
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