Search Results - "Battello, Nadia"

Liver cancer cell lines distinctly mimic the metabolic gene expression pattern of the corresponding human tumours by Nwosu, Zeribe C, Battello, Nadia, Rothley, Melanie, Piorońska, Weronika, Sitek, Barbara, Ebert, Matthias P, Hofmann, Ute, Sleeman, Jonathan, Wölfl, Stefan, Meyer, Christoph, Megger, Dominik A, Dooley, Steven

“…Although metabolism is profoundly altered in human liver cancer, the extent to which experimental models, e.g. cell lines, mimic those alterations is…”
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Transcriptional control of the glucocorticoid receptor: CpG islands, epigenetics and more by Turner, Jonathan D., Alt, Simone R., Cao, Lei, Vernocchi, Sara, Trifonova, Slavena, Battello, Nadia, Muller, Claude P.

“…Glucocorticoid receptor levels are transcriptionally controlled. The variable 5′ region produces multiple splice variants coding a single protein, and is…”
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Severe metabolic alterations in liver cancer lead to ERK pathway activation and drug resistance by Nwosu, Zeribe Chike, Piorońska, Weronika, Battello, Nadia, Zimmer, Andreas David, Dewidar, Bedair, Han, Mei, Pereira, Sharon, Blagojevic, Biljana, Castven, Darko, Charlestin, Verodia, Holenya, Pavlo, Lochead, Julia, De La Torre, Carolina, Gretz, Norbert, Sajjakulnukit, Peter, Zhang, Li, Ward, Matthew H., Marquardt, Jens U., di Magliano, Marina Pasca, Lyssiotis, Costas A., Sleeman, Jonathan, Wölfl, Stefan, Ebert, Matthias Philip, Meyer, Christoph, Hofmann, Ute, Dooley, Steven

“…The extracellular signal-regulated kinase (ERK) pathway regulates cell growth, and is hyper-activated and associated with drug resistance in hepatocellular…”
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The role of HIF-1 in oncostatin M-dependent metabolic reprogramming of hepatic cells by Battello, Nadia, Zimmer, Andreas David, Goebel, Carole, Dong, Xiangyi, Behrmann, Iris, Haan, Claude, Hiller, Karsten, Wegner, Andre

“…Hypoxia and inflammation have been identified as hallmarks of cancer. A majority of hepatocellular carcinomas are preceded by hepatitis B- or C-related chronic…”
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Correction to: Liver cancer cell lines distinctly mimic the metabolic gene expression pattern of the corresponding human tumours by Nwosu, Zeribe C, Battello, Nadia, Rothley, Melanie, Piorońska, Weronika, Sitek, Barbara, Ebert, Matthias P, Hofmann, Ute, Sleeman, Jonathan, Wölfl, Stefan, Meyer, Christoph, Megger, Dominik A, Dooley, Steven

“…In the publication of this article (1), there is an error in Fig. 5b. This has now been updated in the original article (1)…”
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Pro-inflammatory Macrophages Sustain Pyruvate Oxidation through Pyruvate Dehydrogenase for the Synthesis of Itaconate and to Enable Cytokine Expression by Meiser, Johannes, Krämer, Lisa, Sapcariu, Sean C., Battello, Nadia, Ghelfi, Jenny, D'Herouel, Aymeric Fouquier, Skupin, Alexander, Hiller, Karsten

“…Upon stimulation with Th1 cytokines or bacterial lipopolysaccharides, resting macrophages shift their phenotype toward a pro-inflammatory state as part of the…”
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Membrane Glucocorticoid Receptor Activation Induces Proteomic Changes Aligning with Classical Glucocorticoid Effects by Vernocchi, Sara, Battello, Nadia, Schmitz, Stephanie, Revets, Dominique, Billing, Anja M., Turner, Jonathan D., Muller, Claude P.

“…Glucocorticoids exert rapid nongenomic effects by several mechanisms including the activation of a membrane-bound glucocorticoid receptor (mGR). Here, we…”
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ID: 197: Effects of inflammatory cytokines and hypoxia on hepatocytes and HCC cell lines by Zimmer, Andreas David, Battello, Nadia, Hiller, Karsten, Wegner, Andre, Behrmann, Iris, Haan, Claude

“…Hepatocellular carcinoma (HCC), mostly the result of chronic liver diseases, is ranked as the fifth most common cancer world-wide. To find new treatments, a…”
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ID: 197 by Zimmer, Andreas David, Battello, Nadia, Hiller, Karsten, Wegner, Andre, Behrmann, Iris, Haan, Claude

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Bridging the gap between non-targeted stable isotope labeling and metabolic flux analysis by Weindl, Daniel, Cordes, Thekla, Battello, Nadia, Sapcariu, Sean C, Dong, Xiangyi, Wegner, Andre, Hiller, Karsten

“…Metabolism gained increasing interest for the understanding of diseases and to pinpoint therapeutic intervention points. However, classical metabolomics…”
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