Search Results - "Bateman, Raynard L."

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  1. 1

    Human Carbonyl Reductase 1 Is an S-Nitrosoglutathione Reductase by Bateman, Raynard L., Rauh, Daniel, Tavshanjian, Brandon, Shokat, Kevan M.

    Published in The Journal of biological chemistry (19-12-2008)
    “…Human carbonyl reductase 1 (hCBR1) is an NADPH-dependent short chain dehydrogenase/reductase with broad substrate specificity and is thought to be responsible…”
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    Journal Article
  2. 2

    Slow-onset inhibition of fumarylacetoacetate hydrolase by phosphinate mimics of the tetrahedral intermediate: kinetics, crystal structure and pharmacokinetics by Bateman, Raynard L, Ashworth, Justin, Witte, John F, Baker, L-J, Bhanumoorthy, Pullooru, Timm, David E, Hurley, Thomas D, Grompe, Markus, McClard, Ronald W

    Published in Biochemical journal (01-03-2007)
    “…FAH (fumarylacetoacetate hydrolase) catalyses the final step of tyrosine catabolism to produce fumarate and acetoacetate. HT1 (hereditary tyrosinaemia type 1)…”
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    Journal Article
  3. 3

    DNA Replication Is Required To Elicit Cellular Responses to Psoralen-Induced DNA Interstrand Cross-Links by Akkari, Yassmine M. N., Bateman, Raynard L., Reifsteck, Carol A., Olson, Susan B., Grompe, Markus

    Published in Molecular and Cellular Biology (01-11-2000)
    “…Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley…”
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  4. 4

    Mechanistic Inferences from the Crystal Structure of Fumarylacetoacetate Hydrolase with a Bound Phosphorus-based Inhibitor by Bateman, Raynard L., Bhanumoorthy, P., Witte, John F., McClard, Ronald W., Grompe, Markus, Timm, David E.

    Published in The Journal of biological chemistry (04-05-2001)
    “…Fumarylacetoacetate hydrolase (FAH) catalyzes the hydrolytic cleavage of a carbon–carbon bond in fumarylacetoacetate to yield fumarate and acetoacetate as the…”
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    Journal Article
  5. 5

    Preclinical Protocol for in Vivo Selection of Hematopoietic Stem Cells Corrected by Gene Therapy in Fanconi Anemia Group C by Noll, Meenakshi, Bateman, Raynard L., D'Andrea, Alan D., Grompe, Markus

    Published in Molecular therapy (01-01-2001)
    “…Fanconi anemia (FA) is an autosomal recessive disorder characterized by birth defects, increased incidence of malignancy, progressive bone marrow failure, and…”
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  6. 6

    Human Carbonyl Reductase 1 Is an S-Nitrosoglutathione ReductaseS by Bateman, Raynard L., Rauh, Daniel, Tavshanjian, Brandon, Shokat, Kevan M.

    Published in The Journal of biological chemistry (19-12-2008)
    “…Human carbonyl reductase 1 (hCBR1) is an NADPH-dependent short chain dehydrogenase/reductase with broad substrate specificity and is thought to be responsible…”
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    Journal Article
  7. 7

    The 4N Cell Cycle Delay in Fanconi Anemia Reflects Growth Arrest in Late S Phase by Akkari, Yassmine M.N., Bateman, Raynard L., Reifsteck, Carol A., D'Andrea, Alan D., Olson, Susan B., Grompe, Markus

    Published in Molecular genetics and metabolism (01-12-2001)
    “…Fanconi anemia (FA) is a human genetic disorder characterized by hypersensitivity to DNA crosslinking agents. Its cellular phenotypes include increased…”
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    Journal Article
  8. 8

    In vivo selection of wild-type hematopoietic stem cells in a murine model of Fanconi anemia by BATTAILE, K. P, BATEMAN, R. L, MORTIMER, D, MULCAHY, J, RATHBUN, R. K, BAGBY, G, FLEMING, W. H, GROMPE, M

    Published in Blood (15-09-1999)
    “…Fanconi anemia (FA) is an autosomal recessive disorder characterized by birth defects, increased incidence of malignancy, and progressive bone marrow failure…”
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    Journal Article
  9. 9
  10. 10

    In Vivo Selection of Wild-Type Hematopoietic Stem Cells in a Murine Model of Fanconi Anemia by Battaile, Kevin P., Bateman, Raynard L., Mortimer, Derik, Mulcahy, Jean, Rathbun, R. Keaney, Bagby, Grover, Fleming, William H., Grompe, Markus

    Published in Blood (15-09-1999)
    “…Fanconi anemia (FA) is an autosomal recessive disorder characterized by birth defects, increased incidence of malignancy, and progressive bone marrow failure…”
    Get full text
    Journal Article