Search Results - "Ballew, J D"

A Common Polymorphism in SCN5A is Associated with Lone Atrial Fibrillation by Chen, LY, Ballew, JD, Herron, KJ, Rodeheffer, RJ, Olson, TM

“…The cardiac sodium channel (SCN5A) is a target for the treatment of arrhythmias. We hypothesized that vulnerability to atrial fibrillation (AF) could be caused…”
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Cardiac troponin T mutation in familial cardiomyopathy with variable remodeling and restrictive physiology by Menon, SC, Michels, VV, Pellikka, PA, Ballew, JD, Karst, ML, Herron, KJ, Nelson, SM, Rodeheffer, RJ, Olson, TM

“…We identified a unique family with autosomal dominant heart disease variably expressed as restrictive cardiomyopathy (RCM), hypertrophic cardiomyopathy (HCM),…”
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Altered expression of glomerular heat shock protein 27 in experimental nephrotic syndrome by Smoyer, W E, Gupta, A, Mundel, P, Ballew, J D, Welsh, M J

“…Although nephrotic syndrome is a very common kidney disease, little is known about the molecular changes occurring within glomerular capillary loops during…”
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von Willebrand disease in the RIIIS/J mouse is caused by a defect outside of the von Willebrand factor gene by Nichols, W C, Cooney, K A, Mohlke, K L, Ballew, J D, Yang, A, Bruck, M E, Reddington, M, Novak, E K, Swank, R T, Ginsburg, D

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von Willebrand disease in the RIIIS/J mouse is caused by a defect outside of the von Willebrand factor gene by Nichols, W C, Cooney, K A, Mohlke, K L, Ballew, J D, Yang, A, Bruck, M E, Reddington, M, Novak, E K, Swank, R T, Ginsburg, D

“…An animal model for human type I von Willebrand disease (vWD) has been previously described in the inbred mouse strain RIIIS/J. Murine vWD is characterized by…”
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Sodium Channel Mutations and Susceptibility to Heart Failure and Atrial Fibrillation by Olson, Timothy M, Michels, Virginia V, Ballew, Jeffrey D, Reyna, Sandra P, Karst, Margaret L, Herron, Kathleen J, Horton, Steven C, Rodeheffer, Richard J, Anderson, Jeffrey L

“…CONTEXT Dilated cardiomyopathy (DCM), a genetically heterogeneous disorder, causes heart failure and rhythm disturbances. The majority of identified DCM genes…”
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ABCC9 mutations identified in human dilated cardiomyopathy disrupt catalytic K ATP channel gating by Terzic, Andre, Bienengraeber, Martin, Olson, Timothy M, Selivanov, Vitaliy A, Kathmann, Eva C, O'Cochlain, Fearghas, Gao, Fan, Karger, Amy B, Ballew, Jeffrey D, Hodgson, Denice M, Zingman, Leonid V, Pang, Yuan-Ping, Alekseev, Alexey E

“…Stress tolerance of the heart requires high-fidelity metabolic sensing by ATP-sensitive potassium (KATP) channels that adjust membrane potential-dependent…”
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Familial atrial fibrillation is a genetically heterogeneous disorder by Darbar, Dawood, Herron, Kathleen J, Ballew, Jeffrey D, Jahangir, Arshad, Gersh, Bernard J, Shen, Win-K, Hammill, Stephen C, Packer, Douglas L, Olson, Timothy M

“…The aims of this study were to identify and characterize familial cases of atrial fibrillation (AF) in our clinical practice and to determine whether AF is…”
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A common polymorphism in SCN5A is associated with lone atrial fibrillation. Commentary by CARLQUIST, J. F, ANDERSON, J. L, CHEN, L. Y, BALLEW, J. D, HERRON, K. J, RODEHEFFER, R. J, OLSON, T. M

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ABCC9 mutations identified in human dilated cardiomyopathy disrupt catalytic K sub(ATP) channel gating by Bienengraeber, M, Olson, T M, Selivanov, V A, Kathmann, E C, O'Cochlain, F, Gao, F, Karger, AB, Ballew, J D, Hodgson, D M, Zingman, LV, Pang, Y-P, Alekseev, A E, Terzic, A

“…Stress tolerance of the heart requires high-fidelity metabolic sensing by ATP-sensitive potassium (K sub(ATP)) channels that adjust membrane potential-…”
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Procalcitonin Test in the Diagnosis of Bacteremia: A Meta-analysis by Jones, Alan E., MD, Fiechtl, James F., MD, Brown, Michael D., MD, Ballew, Jason J., MD, Kline, Jeffrey A., MD

“…Study objective We seek to evaluate the diagnostic performance of the procalcitonin test for the diagnosis of bacteremia in the emergency department (ED)…”
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von Willebrand disease in the RIIS/J mouse is caused by a defect outside of the von Willebrand factor gene by NICHOLS, W. C, COONEY, K. A, MOHLKE, K. L, BALLEW, J. D, YANG, A, BRUCK, M. E, REDDINGTON, M, NOVAK, E. K, SWANK, R. T, GINSBURG, D

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von Willebrand disease in the RIIIS/J mouse is caused by a defect outside of the von Willebrand factor gene [published erratum appears in Blood 1995 Sep 15;86(6):2461] by Nichols, WC, Cooney, KA, Mohlke, KL, Ballew, JD, Yang, A, Bruck, ME, Reddington, M, Novak, EK, Swank, RT, Ginsburg, D

“…An animal model for human type I von Willebrand disease (vWD) has been previously described in the inbred mouse strain RIIIS/J. Murine vWD is characterized by…”
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