Search Results - "Bakuradze, Ekaterine"

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  1. 1

    Determination of The Properties of Rat Brain Thermostable Protein Complex which Inhibit Cell Proliferation by Dzidziguri, Diana, Modebadze, Irina, Bakuradze, Ekaterine, Mosidze, Giorgi, Berulava, Manana

    Published in Cell journal (Yakhteh) (01-01-2018)
    “…Cell proliferation is known to be controlled by many networks of regulatory proteins. These multiple and complicated mechanisms of control are still being…”
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    Journal Article
  2. 2

    The influence of endogenous protein complex on morpho‐functional activity of bone marrow cells (616.3) by Bakuradze, Ekaterine, Modebadze, Irina, Mosidze, Giorgi, Peradze, Eka, Dzidziguri, Diana

    Published in The FASEB journal (01-04-2014)
    “…Endogenous regulators of proliferation are considered as a main player of cell‐cycle regulatory system. Specific actions (stimulation or inhibition) on the…”
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    Journal Article
  3. 3

    Adaptive growth features of rat cholestatic liver by Modebadze, Irina, Bakuradze, Ekaterine, Ninua, Levan, Dekanoidze, Davit, Dzidziguri, Diana

    Published in The FASEB journal (01-04-2011)
    “…It is known that along with proliferation a high profile is given to a cell hypertrophy and polyploidy in restoration of structure and function of various…”
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    Journal Article
  4. 4

    The adaptation mechanisms of liver in combined pathologies by Dzidziguri, Diana, Bakuradze, Ekaterine, Modebadze, Irina, Ninua, Levan, Dekanoidze, Davit, Rusishvili, Levan

    Published in The FASEB journal (01-04-2010)
    “…The great potential of liver restoration has been described in different liver pathologies. There are various forms of liver diseases. Hence, comparative study…”
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    Journal Article
  5. 5

    The comparative study of endogenous growth inhibiting factors of pro‐ and eukaryotic cells by Dzidziguri, Diana, Rukhadze, Marina, Bakuradze, Ekaterine, Modebadze, Irina, Gorozia, Ilia, Abashidze, Vera

    Published in The FASEB journal (01-04-2009)
    “…Abstract only Tumour cells are often resistant to signals which inhibit growth of normal cells. Avoiding such control mechanisms may contribute to malignant…”
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