Search Results - "Bösche, Leif I"
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Initial Experience With Novel Oral Anticoagulants During the First 45 Days After Left Atrial Appendage Closure With the Watchman Device
Published in Clinical cardiology (Mahwah, N.J.) (01-12-2015)“…ABSTRACT Background The use of oral anticoagulation or dual antiplatelet therapy (DAPT) is recommended within the first 45 days after left atrial appendage…”
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Correlation Between Total Atrial Conduction Time Estimated via Tissue Doppler Imaging (PA-TDI Interval), Structural Atrial Remodeling and New-Onset of Atrial Fibrillation After Cardiac Surgery
Published in Journal of cardiovascular electrophysiology (01-06-2013)“…Atrial Conduction Time Background Recent studies identified total atrial conduction time (TACT) as an independent and powerful predictor of new‐onset atrial…”
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Emoji-like structure in the right atrium: identified as blood cyst and removed by interventional venous drainage cannula
Published in European heart journal (01-01-2023)Get full text
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Generation of a constitutive Na+-dependent inward-rectifier current in rat adult atrial myocytes by overexpression of Kir3.4
Published in The Journal of physiology (15-11-2007)“…Apart from gating by interaction with βγ subunits from heterotrimeric G proteins upon stimulation of appropriate receptors, Kir.3 channels have been shown to…”
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Acute desensitization of GIRK current in rat atrial myocytes is related to K+ current flow
Published in The Journal of physiology (01-12-2004)“…We have investigated the acute desensitization of acetylcholine-activated GIRK current ( I K(ACh) ) in cultured adult rat atrial myocytes. Acute…”
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Termination of Ventricular Tachycardia by Far-Field Stimulation in Humans: A Feasibility Study
Published in Pacing and clinical electrophysiology (01-12-2010)“…Background: Although a low‐energy cardioversion (LEC) shock from an implantable cardioverter‐defibrillator (ICD) can terminate ventricular tachycardia (VT), it…”
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Gene silencing in adult rat cardiac myocytes in vitro by adenovirus-mediated RNA interference
Published in Journal of muscle research and cell motility (01-05-2006)“…RNA interference (RNAi) by short double stranded RNA (siRNA) represents an efficient and frequently used tool for gene silencing to study gene function…”
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G Protein-Independent Inhibition of GIRK Current by Adenosine in Rat Atrial Myocytes Overexpressing A1 Receptors after Adenovirus-Mediated Gene Transfer
Published in The Journal of physiology (01-08-2003)“…G protein-activated inwardly rectifying K + (GIRK) channels, important regulators of membrane excitability in the heart and central nervous system, are…”
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Transfection with 5-HT1A receptor gene and antisense directed against muscarinic M2 receptors reveal a mutual influence between Gi/o-coupled receptors in rat atrial myocytes
Published in Journal of molecular and cellular cardiology (01-01-2003)“…A recently described reduction in sensitivity of G protein-activated inward-rectifying K(+) (GIRK) channels to stimulation of muscarinic M(2) receptors…”
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G Protein-Activated (GIRK) Current in Rat Ventricular Myocytes is Masked by Constitutive Inward Rectifier Current (IK1)
Published in Cellular physiology and biochemistry (01-01-2008)“…Inwardly-rectifying K + channel subunits are not homogenously expressed in different cardiac tissues. In ventricular myocytes (VM) the background…”
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G Protein‐Independent Inhibition of GIRK Current by Adenosine in Rat Atrial Myocytes Overexpressing A 1 Receptors after Adenovirus‐Mediated Gene Transfer
Published in The Journal of physiology (01-08-2003)“…G protein‐activated inwardly rectifying K + (GIRK) channels, important regulators of membrane excitability in the heart and central nervous system, are…”
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12
Transfection with 5-HT 1A receptor gene and antisense directed against muscarinic M 2 receptors reveal a mutual influence between G i/o-coupled receptors in rat atrial myocytes
Published in Journal of molecular and cellular cardiology (2003)“…A recently described reduction in sensitivity of G protein-activated inward-rectifying K + (GIRK) channels to stimulation of muscarinic M 2 receptors (M 2AChR)…”
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