Search Results - "Börzsönyi, B."

W128 PATHOLOGICAL FETOMATERNAL GLUCOCORTICOID METABOLISM AS A CAUSE OF PRETERM DELIVERY; THE ETIOLOGICAL ROLE OF THE PLACENTAL 11β-HYDROXYSTEROID DEHIDROGENASE 2 GENE by Szentpéteri, I., Börzsönyi, B., Demendi, C., Nagy, Z., Joó, J.G.

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W154 PLACENTAL GENE EXPRESSION OF BAX AND BCL-2 GENES; APOPTOSIS AS A POTENTIAL CAUSE OF INTRAUTERINE GROWTH RESTRICTION by Börzsönyi, B., Demendi, C., Nagy, Z., Pajor, A., Rigo, J., Szentpéteri, I., Joó, J.G.

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O345 ETIOLOGICAL ROLE OF INSULIN-LIKE GROWTH FACTOR 1 (IGF-1); INSULIN-LIKE GROWTH FACTOR 2 (IGF-2) AND INSULIN-LIKE GROWTH FACTOR BINDING PROTEIN 3 (IGFBP-3) GENE IN THE BACKGROUND OF INTRAUTERINE RESTRICTION; ANALYSIS OF PLACENTAL GENE EXPRESSION PATTERN by Joó, J.G., Börzsönyi, B., Demendi, C., Nagy, Z., Szentpéteri, I., Pajor, A., Rigo, J.

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W132 THE ROLE OF IGF-1; IGF-2 AND IGFP-3 GENES IN THE ETIOLOGY OF PRETERM DELIVERIES; PLACENTAL GENE EXPRESSION ANALYSIS by Demendi, C., Börzsönyi, B., Nagy, Z., Szentpetéri, I., Pajor, A., Rigo, J., Joó, J.G.

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W155 IMPAIRED FETOMATERNAL GLUCOCORTICOID METABOLISM AS AN ETIOLOGICAL FACTOR OF INTRAUTERINE GROWTH RESTRICTION; PLACENTAL EXPRESSION OF 11β-HYDROXYSTEROID DEHYDROGENASE 2 by Börzsönyi, B., Demendi, C., Nagy, Z., Szentpéteri, I., Pajor, A., Rigo, J., Joó, J.G.

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W136 APOPTOSIS AND PRETERM DELIVERY; PLACENTAL GENE EXPRESSION OF BAX- AND BCL-2 GENES by Demendi, C., Börzsönyi, B., Nagy, Z., Szentpéteri, I., Pajor, A., Rigo, J., Joó, J.G.

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Luteal function as the main determinant of pregnancy outcome: successful prevention of spontaneous abortion, prematurity and IUGR by Siklósi, G, Acs, N, Demendi, C, Börzsönyi, B, Gimes, G, Bakos, L, Olajos, F, Marcsek, Z

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Emergency contraception: Experiences with fertilan by Csömör, S., Börzsönyi, B., Kazy, Z., Lukácsi, L.

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Current concepts in the genetic diagnostics of rheumatoid arthritis by Nagy, Zsolt B, Csanád, Mónika, Tóth, Katalin, Börzsönyi, Balázs, Demendi, Csaba, Rigó, Jr, János, Joó, József Gábor

“…Rheumatoid arthritis (RA) is a systemic, chronic and inflammatory disease of unknown etiology. HLA-DRB1 and PTPN22 1858T gene variants are risk factors of RA,…”
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