Search Results - "Aymeric, Laetitia"
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Significance of Streptococcus gallolyticus subsp. gallolyticus Association With Colorectal Cancer
Published in Frontiers in microbiology (03-04-2018)“…subsp. (formerly known as type I) is the main causative agent of septicemia and infective endocarditis (IE) in elderly and immunocompromised persons. It…”
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2
Tumor Cell Death and ATP Release Prime Dendritic Cells and Efficient Anticancer Immunity
Published in Cancer research (Chicago, Ill.) (01-02-2010)“…By destroying tumor cells, conventional anticancer therapies may stimulate the host immune system to eliminate residual disease. Anthracyclines, oxaliplatin,…”
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3
Anticancer Chemotherapy-Induced Intratumoral Recruitment and Differentiation of Antigen-Presenting Cells
Published in Immunity (Cambridge, Mass.) (18-04-2013)“…The therapeutic efficacy of anthracyclines relies on antitumor immune responses elicited by dying cancer cells. How chemotherapy-induced cell death leads to…”
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4
IL-18 Induces PD-1: Dependent Immunosuppression in Cancer
Published in Cancer research (Chicago, Ill.) (15-08-2011)“…Immunosuppressive cytokines subvert innate and adaptive immune responses during cancer progression. The inflammatory cytokine interleukin-18 (IL-18) is known…”
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5
Late-Stage Glioma Is Associated with Deleterious Alteration of Gut Bacterial Metabolites in Mice
Published in Metabolites (25-03-2022)“…Brain-gut axis refers to the bidirectional functional connection between the brain and the gut, which sustains vital functions for vertebrates. This connection…”
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6
The dendritic cell–tumor cross-talk in cancer
Published in Current opinion in immunology (01-02-2011)“…The question as to whether the tumor grows because of or despite the host immune system is being progressively addressed with refined technology, gene…”
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7
Chemotherapy and radiotherapy: Cryptic anticancer vaccines
Published in Seminars in immunology (01-06-2010)“…Abstract An attractive, yet hitherto unproven concept predicts that the promotion of tumor regression should elicit the host's immune response against residual…”
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8
Psychological stress induces an increase in cholinergic enteric neuromuscular pathways mediated by glucocorticoid receptors
Published in Frontiers in neuroscience (14-02-2023)“…Repeated acute stress (RASt) is known to be associated with gastrointestinal dysfunctions. However, the mechanisms underlying these effects have not yet been…”
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9
Cancer-Induced Immunosuppression: IL-18-Elicited Immunoablative NK Cells
Published in Cancer research (Chicago, Ill.) (01-06-2012)“…During cancer development, a number of regulatory cell subsets and immunosuppressive cytokines subvert adaptive immune responses. Although it has been shown…”
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10
How to improve the immunogenicity of chemotherapy and radiotherapy
Published in Cancer and metastasis reviews (01-03-2011)“…Chemotherapy or radiotherapy could induce various tumor cell death modalities, releasing tumor-derived antigen as well as danger signals that could either be…”
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11
Fecal Supernatant from Adult with Autism Spectrum Disorder Alters Digestive Functions, Intestinal Epithelial Barrier, and Enteric Nervous System
Published in Microorganisms (Basel) (13-08-2021)“…Autism Spectrum Disorders (ASDs) are neurodevelopmental disorders defined by impaired social interactions and communication with repetitive behaviors,…”
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12
Colorectal cancer specific conditions promote Streptococcus gallolyticus gut colonization
Published in Proceedings of the National Academy of Sciences - PNAS (09-01-2018)“…Colonization by Streptococcus gallolyticus subsp. gallolyticus (SGG) is strongly associated with the occurrence of colorectal cancer (CRC). However, the…”
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13
Activation of the NLRP3 inflammasome in dendritic cells induces IL-1β-dependent adaptive immunity against tumors
Published in Nature medicine (01-10-2009)“…Dying tumor cells release ATP, which activates the NLRP3 inflammasome in dendritic cells, enabling the secretion of interleukin-1β and the subsequent priming…”
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14
Contribution of IL-17–producing γδ T cells to the efficacy of anticancer chemotherapy
Published in The Journal of experimental medicine (14-03-2011)“…By triggering immunogenic cell death, some anticancer compounds, including anthracyclines and oxaliplatin, elicit tumor-specific, interferon-γ–producing CD8+…”
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15
Contribution of IL-17-producing gamma delta T cells to the efficacy of anticancer chemotherapy
Published in The Journal of experimental medicine (14-03-2011)“…By triggering immunogenic cell death, some anticancer compounds, including anthracyclines and oxaliplatin, elicit tumor-specific, interferon-γ-producing CD8(+)…”
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16
Streptococcus gallolyticus Pil3 Pilus Is Required for Adhesion to Colonic Mucus and for Colonization of Mouse Distal Colon
Published in The Journal of infectious diseases (15-11-2015)“…Streptococcus gallolyticus is an increasing cause of bacteremia and infective endocarditis in the elderly. Several epidemiological studies have associated the…”
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17
CCL2/CCR2-Dependent Recruitment of Functional Antigen-Presenting Cells into Tumors upon Chemotherapy
Published in Cancer research (Chicago, Ill.) (15-01-2014)“…The therapeutic efficacy of anthracyclines relies, at least partially, on the induction of a dendritic cell- and T-lymphocyte-dependent anticancer immune…”
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18
Adhesion of Gastric Cancer Cells to the Enteric Nervous System: Comparison between the Intestinal Type and Diffuse Type of Gastric Cancer
Published in Cancers (06-07-2022)“…Background: Gastric cancer (GC) is the third leading cause of cancer-related deaths worldwide. The enteric nervous system (ENS) has been suggested to be…”
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19
Cell-death-associated molecular patterns as determinants of cancer immunogenicity
Published in Antioxidants & redox signaling (01-03-2014)“…Accumulating evidence indicates that the success of some anticancer treatments (select chemotherapies or radiotherapy or trastuzumab) could be related to the…”
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20
Desirable cell death during anticancer chemotherapy
Published in Annals of the New York Academy of Sciences (01-10-2010)“…The concept of immunogenic chemotherapy that has recently emerged relies upon the capacity of a cytotoxic compound to trigger a cell‐death modality. This…”
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