Search Results - "Atla, G."

Genetic architecture of oral glucose-stimulated insulin release provides biological insights into type 2 diabetes aetiology by Madsen, A. L., Bonàs-Guarch, S., Gheibi, S., Prasad, R., Vangipurapu, J., Ahuja, V., Cataldo, L. R., Dwivedi, O., Hatem, G., Atla, G., Guindo-Martínez, M., Jørgensen, A. M., Jonsson, A. E., Miguel-Escalada, I., Hassan, S., Linneberg, A., Ahluwalia, Tarunveer S., Drivsholm, T., Pedersen, O., Sørensen, T. I. A., Astrup, A., Witte, D., Damm, P., Clausen, T. D., Mathiesen, E., Pers, T. H., Loos, R. J. F., Hakaste, L., Fex, M., Grarup, N., Tuomi, T., Laakso, M., Mulder, H., Ferrer, J., Hansen, T.

“…The genetics of β-cell function (BCF) offer valuable insights into the aetiology of type 2 diabetes (T2D) 1 , 2 . Previous studies have expanded the catalogue…”
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REST is a major negative regulator of endocrine differentiation during pancreas organogenesis by Rovira, Meritxell, Atla, Goutham, Maestro, Miguel Angel, Grau, Vane, García-Hurtado, Javier, Maqueda, Maria, Mosquera, Jose Luis, Yamada, Yasuhiro, Kerr-Conte, Julie, Pattou, Francois, Ferrer, Jorge

“…Multiple transcription factors have been shown to promote pancreatic β-cell differentiation, yet much less is known about negative regulators. Earlier…”
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