Search Results - "Ascah, A."

Citrulline as a Biomarker for Gastrointestinal-Acute Radiation Syndrome: Species Differences and Experimental Condition Effects by Bujold, K., Hauer-Jensen, M., Donini, O., Rumage, A., Hartman, D., Hendrickson, H. P., Stamatopoulos, J., Naraghi, H., Pouliot, M., Ascah, A., Sebastian, M., Pugsley, M. K., Wong, K., Authier, S.

“…Animal models of hematopoietic and gastrointestinal acute radiation syndromes (ARS) have been characterized to develop medical countermeasures. Acute…”
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Intensity‐dependent activation of intracellular signalling pathways in skeletal muscle: role of fibre type recruitment during exercise by Godin, R., Ascah, A., Daussin, F. N.

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Rat cardiovascular telemetry: Marginal distribution applied to positive control drugs by Accardi, M.V., Troncy, E., Abtout, S., Ascah, A., Maghezzi, S., Authier, S.

“…Cardiovascular effects are considered frequent during drug safety testing. This investigation aimed to characterize the pharmacological response of the…”
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Data visualization in an era of multi-ion channel inhibition strategies by Huang, H., Accardi, M.V., Bujold, K., Ascah, A., Abtout, S., Pouliot, M., Authier, S.

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Intensity-dependent activation of intracellular signalling pathways in skeletal muscle: role of fibre type recruitment during exercise: Journal Club by Godin, R., Ascah, A., Daussin, F. N.

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Exercise training induces respiratory substrate-specific decrease in Ca2+-induced permeability transition pore opening in heart mitochondria by Marcil, Mariannick, Bourduas, Karine, Ascah, Alexis, Burelle, Yan

“…The purpose of this study was to determine whether regular exercise (treadmill running, 10 wk) alters the susceptibility of rat isolated heart mitochondria to…”
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Compensated volume overload increases the vulnerability of heart mitochondria without affecting their functions in the absence of stress by Marcil, Mariannick, Ascah, Alexis, Matas, Jimmy, Bélanger, Sonia, Deschepper, Christian F., Burelle, Yan

“…Although mitochondrial dysfunction has often been associated to heart failure, it has been suggested that it may represent only a late phenomenon in the…”
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A52. Chronic volume overload increases the vulnerability of cardiac mitochondria without affecting their basal functions by Marcil, M., Ascah, A., Bélanger, S., Deschepper, C.F., Burelle, Y.

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