Search Results - "Armstead, WM"
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Young Age as a Risk Factor for Impaired Cerebral Autoregulation after Moderate to Severe Pediatric Traumatic Brain Injury
Published in Anesthesiology (Philadelphia) (01-04-2008)“…Little is known about age and cerebral autoregulation in children with traumatic brain injury (TBI). The authors compared cerebral autoregulation between young…”
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uPA impairs cerebrovasodilation after hypoxia/ischemia through LRP and ERK MAPK
Published in Brain research (22-09-2008)“…Abstract We have previously observed that soluble urokinase plasminogn activator receptor (suPAR) prevents impairment of cerebrovasodilation induced by…”
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3
Nociceptin/orphanin FQ dilates pial arteries by K ATP and K ca channel activation
Published in Brain research (24-07-1999)“…Nociceptin/orphanin FQ (NOC/oFQ) is a recently discovered endogenous ligand for the opioid like receptor, ORL-1. In the piglet, cGMP activates the ATP…”
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4
Vasopressin induced cyclooxygenase dependent superoxide generation contributes to K super(+) channel function impairment after brain injury
Published in Brain research (10-08-2001)“…This study determined if vasopressin generates superoxide anion (O super(-) sub(2)) in a cyclooxygenase dependent manner and if such production contributes to…”
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5
NOC/oFQ activates ERK and JNK but not p38 MAPK to impair prostaglandin cerebrovasodilation after brain injury
Published in Brain research (23-08-2005)“…Fluid percussion brain injury (FPI) elevates the CSF concentration of the opioid nociceptin/orphanin FQ (NOC/oFQ), which contributes to impairment of pial…”
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Role of endothelin-1 in age-dependent cerebrovascular hypotensive responses after brain injury
Published in The American journal of physiology (01-11-1999)“…This study was designed to compare the effect of fluid percussion brain injury (FPI) on the hypotensive cerebrovascular response in newborn and juvenile pigs…”
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Protein tyrosine kinase and mitogen-activated protein kinase activation contribute to K ATP and K ca channel impairment after brain injury
Published in Brain research (12-07-2002)“…Previous studies have observed that pial artery dilation to activators of the ATP sensitive K (K ATP) and calcium sensitive K (K ca) channel was blunted…”
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Endothelins and the role of endothelin antagonists in the management of posttraumatic vasospasm
Published in Current pharmaceutical design (01-07-2004)“…Increasing evidence implicates endothelin in the pathophysiological development of cerebral vasospasm. This review summarizes background topics such as the…”
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Plasminogen activators contribute to impairment of hypercapnic and hypotensive cerebrovasodilation after cerebral hypoxia/ischemia in the newborn pig
Published in Stroke (1970) (01-10-2005)“…Babies are frequently exposed to hypoxia and ischemia during the perinatal period as a result of stroke or problems with delivery or respiratory management…”
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Vasopressin-induced protein kinase C-dependent superoxide generation contributes to ATP-sensitive potassium channel but not calcium-sensitive potassium channel function impairment after brain injury
Published in Stroke (1970) (01-06-2001)“…Pial artery dilation in response to activators of the ATP-sensitive K(+) (K(ATP)) and calcium-sensitive K(+) (K(Ca)) channels is impaired after fluid…”
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11
G protein activation elicits cerebrovasodilation through interaction with K ATP and K Ca channels
Published in Brain research (13-12-2002)“…This study was designed to characterize the vascular activity of two activators of a pertussis toxin-sensitive G protein, mastoparan and mastoparan-7, on pial…”
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12
PTK, ERK and p38 MAPK contribute to impaired NMDA-induced vasodilation after brain injury
Published in European journal of pharmacology (08-08-2003)“…N-Methyl- d-aspartate (NMDA)-induced pial artery dilation is reversed to vasoconstriction following fluid percussion brain injury (FPI). This study…”
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13
Superoxide generation links protein kinase C activation to impaired ATP-sensitive K+ channel function after brain injury
Published in Stroke (1970) (01-01-1999)“…BACKGROUND AND PURPOSE--Endothelin-1, in concentrations similar to that present in cerebrospinal fluid after fluid percussion brain injury (FPI), increases…”
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Role of ATP-sensitive K+ channels in cGMP-mediated pial artery vasodilation
Published in The American journal of physiology (01-02-1996)“…The present study was designed to investigate the role of ATP-sensitive K+ channels in guanosine 3',5'-cyclic monophosphate (cGMP)-mediated pial artery…”
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15
Vasopressin induced cyclooxygenase dependent superoxide generation contributes to K + channel function impairment after brain injury
Published in Brain research (10-08-2001)“…This study determined if vasopressin generates superoxide anion (O − 2) in a cyclooxygenase dependent manner and if such production contributes to impairment…”
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16
Protein tyrosine kinase and mitogen-activated protein kinase activation contribute to KATP and Kca channel impairment after brain injury
Published in Brain research (12-07-2002)“…Previous studies have observed that pial artery dilation to activators of the ATP sensitive K (KATP) and calcium sensitive K (Kca) channel was blunted…”
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17
Nociceptin/orphanin FQ dilates pial arteries by K sub(ATP) and K sub(ca) channel activation
Published in Brain research (24-07-1999)“…Nociceptin/orphanin FQ (NOC/oFQ) is a recently discovered endogenous ligand for the opioid like receptor, ORL-1. In the piglet, cGMP activates the ATP…”
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Journal Article -
18
Protein kinase C activation generates superoxide and contributes to impairment of cerebrovasodilation induced by G protein activation after brain injury
Published in Brain research (09-05-2003)“…Previous studies have observed that activation of protein kinase C (PKC) contributes to generation of superoxide anion (O − 2) after fluid percussion brain…”
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Relationship between NOC/oFQ, dynorphin, and COX-2 activation in impaired NMDA cerebrovasodilation after brain injury
Published in Journal of neurotrauma (01-08-2002)“…Previous studies have observed that the recently described endogenous opioid, nociceptin/orphanin FQ (NOC/oFQ), contributes to impairment of…”
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20
G protein activation elicits cerebrovasodilation through interaction with KATP and KCa channels
Published in Brain research (13-12-2002)“…This study was designed to characterize the vascular activity of two activators of a pertussis toxin-sensitive G protein, mastoparan and mastoparan-7, on pial…”
Get full text
Journal Article