Search Results - "Armstead, WM"

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  1. 1

    Young Age as a Risk Factor for Impaired Cerebral Autoregulation after Moderate to Severe Pediatric Traumatic Brain Injury by FREEMAN, Serena S, UDOMPHORN, Yuthana, ARMSTEAD, William M, FISK, Dana M, VAVILALA, Monica S

    Published in Anesthesiology (Philadelphia) (01-04-2008)
    “…Little is known about age and cerebral autoregulation in children with traumatic brain injury (TBI). The authors compared cerebral autoregulation between young…”
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    Journal Article
  2. 2

    uPA impairs cerebrovasodilation after hypoxia/ischemia through LRP and ERK MAPK by Armstead, William M, Cines, Douglas B, Bdeir, Khalil, Kulikovskaya, Irina, Stein, Sherman C, Higazi, Abd Al-Roof

    Published in Brain research (22-09-2008)
    “…Abstract We have previously observed that soluble urokinase plasminogn activator receptor (suPAR) prevents impairment of cerebrovasodilation induced by…”
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    Journal Article
  3. 3

    Nociceptin/orphanin FQ dilates pial arteries by K ATP and K ca channel activation by Armstead, William M

    Published in Brain research (24-07-1999)
    “…Nociceptin/orphanin FQ (NOC/oFQ) is a recently discovered endogenous ligand for the opioid like receptor, ORL-1. In the piglet, cGMP activates the ATP…”
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    Journal Article
  4. 4

    Vasopressin induced cyclooxygenase dependent superoxide generation contributes to K super(+) channel function impairment after brain injury by Armstead, WM

    Published in Brain research (10-08-2001)
    “…This study determined if vasopressin generates superoxide anion (O super(-) sub(2)) in a cyclooxygenase dependent manner and if such production contributes to…”
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    Journal Article
  5. 5

    NOC/oFQ activates ERK and JNK but not p38 MAPK to impair prostaglandin cerebrovasodilation after brain injury by Ross, John, Armstead, William M.

    Published in Brain research (23-08-2005)
    “…Fluid percussion brain injury (FPI) elevates the CSF concentration of the opioid nociceptin/orphanin FQ (NOC/oFQ), which contributes to impairment of pial…”
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  6. 6

    Role of endothelin-1 in age-dependent cerebrovascular hypotensive responses after brain injury by Armstead, W M

    Published in The American journal of physiology (01-11-1999)
    “…This study was designed to compare the effect of fluid percussion brain injury (FPI) on the hypotensive cerebrovascular response in newborn and juvenile pigs…”
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  7. 7

    Protein tyrosine kinase and mitogen-activated protein kinase activation contribute to K ATP and K ca channel impairment after brain injury by Armstead, William M

    Published in Brain research (12-07-2002)
    “…Previous studies have observed that pial artery dilation to activators of the ATP sensitive K (K ATP) and calcium sensitive K (K ca) channel was blunted…”
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    Journal Article
  8. 8

    Endothelins and the role of endothelin antagonists in the management of posttraumatic vasospasm by Armstead, W M

    Published in Current pharmaceutical design (01-07-2004)
    “…Increasing evidence implicates endothelin in the pathophysiological development of cerebral vasospasm. This review summarizes background topics such as the…”
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    Journal Article
  9. 9

    Plasminogen activators contribute to impairment of hypercapnic and hypotensive cerebrovasodilation after cerebral hypoxia/ischemia in the newborn pig by ARMSTEAD, William M, CINES, Douglas B, HIGAZI, Abd Al-Roof

    Published in Stroke (1970) (01-10-2005)
    “…Babies are frequently exposed to hypoxia and ischemia during the perinatal period as a result of stroke or problems with delivery or respiratory management…”
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    Journal Article
  10. 10

    Vasopressin-induced protein kinase C-dependent superoxide generation contributes to ATP-sensitive potassium channel but not calcium-sensitive potassium channel function impairment after brain injury by ARMSTEAD, William M

    Published in Stroke (1970) (01-06-2001)
    “…Pial artery dilation in response to activators of the ATP-sensitive K(+) (K(ATP)) and calcium-sensitive K(+) (K(Ca)) channels is impaired after fluid…”
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    Journal Article
  11. 11

    G protein activation elicits cerebrovasodilation through interaction with K ATP and K Ca channels by Armstead, William M

    Published in Brain research (13-12-2002)
    “…This study was designed to characterize the vascular activity of two activators of a pertussis toxin-sensitive G protein, mastoparan and mastoparan-7, on pial…”
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    Journal Article
  12. 12

    PTK, ERK and p38 MAPK contribute to impaired NMDA-induced vasodilation after brain injury by Armstead, William M.

    Published in European journal of pharmacology (08-08-2003)
    “…N-Methyl- d-aspartate (NMDA)-induced pial artery dilation is reversed to vasoconstriction following fluid percussion brain injury (FPI). This study…”
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    Journal Article
  13. 13

    Superoxide generation links protein kinase C activation to impaired ATP-sensitive K+ channel function after brain injury by Armstead, W M

    Published in Stroke (1970) (01-01-1999)
    “…BACKGROUND AND PURPOSE--Endothelin-1, in concentrations similar to that present in cerebrospinal fluid after fluid percussion brain injury (FPI), increases…”
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    Journal Article
  14. 14

    Role of ATP-sensitive K+ channels in cGMP-mediated pial artery vasodilation by Armstead, W M

    Published in The American journal of physiology (01-02-1996)
    “…The present study was designed to investigate the role of ATP-sensitive K+ channels in guanosine 3',5'-cyclic monophosphate (cGMP)-mediated pial artery…”
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    Journal Article
  15. 15

    Vasopressin induced cyclooxygenase dependent superoxide generation contributes to K + channel function impairment after brain injury by Armstead, William M

    Published in Brain research (10-08-2001)
    “…This study determined if vasopressin generates superoxide anion (O − 2) in a cyclooxygenase dependent manner and if such production contributes to impairment…”
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    Journal Article
  16. 16

    Protein tyrosine kinase and mitogen-activated protein kinase activation contribute to KATP and Kca channel impairment after brain injury by ARMSTEAD, William M

    Published in Brain research (12-07-2002)
    “…Previous studies have observed that pial artery dilation to activators of the ATP sensitive K (KATP) and calcium sensitive K (Kca) channel was blunted…”
    Get full text
    Journal Article
  17. 17

    Nociceptin/orphanin FQ dilates pial arteries by K sub(ATP) and K sub(ca) channel activation by Armstead, WM

    Published in Brain research (24-07-1999)
    “…Nociceptin/orphanin FQ (NOC/oFQ) is a recently discovered endogenous ligand for the opioid like receptor, ORL-1. In the piglet, cGMP activates the ATP…”
    Get full text
    Journal Article
  18. 18

    Protein kinase C activation generates superoxide and contributes to impairment of cerebrovasodilation induced by G protein activation after brain injury by Armstead, William M.

    Published in Brain research (09-05-2003)
    “…Previous studies have observed that activation of protein kinase C (PKC) contributes to generation of superoxide anion (O − 2) after fluid percussion brain…”
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    Journal Article
  19. 19

    Relationship between NOC/oFQ, dynorphin, and COX-2 activation in impaired NMDA cerebrovasodilation after brain injury by Kulkarni, Miriam, Armstead, William M

    Published in Journal of neurotrauma (01-08-2002)
    “…Previous studies have observed that the recently described endogenous opioid, nociceptin/orphanin FQ (NOC/oFQ), contributes to impairment of…”
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    Journal Article
  20. 20

    G protein activation elicits cerebrovasodilation through interaction with KATP and KCa channels by ARMSTEAD, William M

    Published in Brain research (13-12-2002)
    “…This study was designed to characterize the vascular activity of two activators of a pertussis toxin-sensitive G protein, mastoparan and mastoparan-7, on pial…”
    Get full text
    Journal Article