Search Results - "Aranyos, Alek"

Berberine potentiates liver inflammation and fibrosis in the PIZ hAAT transgenic murine model by Lu, Yuanqing, Mohammad, Naweed S, Lee, Jungnam, Aranyos, Alek M, Serban, Karina A, Brantly, Mark L

“…Alpha-1 antitrypsin deficiency (AATD) is an inherited disease, the common variant caused by a Pi*Z mutation in the SERPINA1 gene. Pi*Z AAT increases the risk…”
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Characterization of hepatic inflammatory changes in a C57BL/6J mouse model of alpha1-antitrypsin deficiency by Khodayari, Nazli, Oshins, Regina, Aranyos, Alek M, Duarte, Sergio, Mostofizadeh, Sayedamin, Lu, Yuanqing, Brantly, Mark

“…Alpha-1 antitrypsin deficiency (AATD) is a genetic disease caused by a hepatic accumulation of mutant alpha-1 antitrypsin (ZAAT). Individuals with AATD are…”
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Alpha-defensins inhibit ERK/STAT3 signaling during monocyte-macrophage differentiation and impede macrophage function by Lee, Jungnam, Mohammad, Naweed, Lu, Yuanqing, Oshins, Regina, Aranyos, Alek, Brantly, Mark

“…Alpha-1-antitrypsin deficiency (AATD) is a genetic disorder associated with a 5-tenfold decrease in lung levels of alpha-1-antitrypsin (AAT) and an increased…”
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The Mechanism of Mitochondrial Injury in Alpha-1 Antitrypsin Deficiency Mediated Liver Disease by Khodayari, Nazli, Wang, Rejean L, Oshins, Regina, Lu, Yuanqing, Millett, Michael, Aranyos, Alek M, Mostofizadeh, Sayedamin, Scindia, Yogesh, Flagg, Tammy O, Brantly, Mark

“…Alpha-1 antitrypsin deficiency (AATD) is caused by a single mutation in the SERPINA1 gene, which culminates in the accumulation of misfolded alpha-1…”
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Utilization of Feline ELISpot to Evaluate the Immunogenicity of a T Cell-Based FIV MAP Vaccine by Sahay, Bikash, Aranyos, Alek M, McAvoy, Andrew, Yamamoto, Janet K

“…The prototype and the commercial dual-subtype feline immunodeficiency virus (FIV) vaccines conferred protection against homologous FIV strains as well as…”
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Immunogenicity and Efficacy of a Novel Multi-Antigenic Peptide Vaccine Based on Cross-Reactivity between Feline and Human Immunodeficiency Viruses by Sahay, Bikash, Aranyos, Alek M, Mishra, Meerambika, McAvoy, Andrew C, Martin, Marcus M, Pu, Riuyu, Shiomitsu, Sayaka, Shiomitsu, Keijiro, Dark, Michael J, Sanou, Missa P, Roff, Shannon R, Rathore, Mobeen H, Yamamoto, Janet K

“…For the development of an effective HIV-1 vaccine, evolutionarily conserved epitopes between feline and human immunodeficiency viruses (FIV and HIV-1) were…”
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The unfolded protein response to PIZ alpha‐1 antitrypsin in human hepatocellular and murine models by Lu, Yuanqing, Wang, Liqun R., Lee, Jungnam, Mohammad, Naweed S., Aranyos, Alek M., Gould, Calvin, Khodayari, Nazli, Oshins, Regina A., Moneypenny, Craig G., Brantly, Mark L.

“…Alpha‐1 antitrypsin (AAT) deficiency (AATD) is an inherited disease caused by mutations in the serpin family A member 1 (SERPINA1, also known as AAT) gene. The…”
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An initial examination of the potential role of T-cell immunity in protection against feline immunodeficiency virus (FIV) infection by Aranyos, Alek M, Roff, Shannon R, Pu, Ruiyu, Owen, Jennifer L, Coleman, James K, Yamamoto, Janet K

“…Highlights • 23/33 recipients of adoptive T-cell transfer from vaccinated donors were protected. • 81% (17/21) protection was against homologous FIV (vaccine…”
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Sirtuin3 promotes the degradation of hepatic Z alpha-1 antitrypsin through lipophagy by Poole, Brittney, Oshins, Regina, Huo, Zhiguang, Aranyos, Alek, West, Jesse, Duarte, Sergio, Clark, Virginia C, Beduschi, Thiago, Zarrinpar, Ali, Brantly, Mark, Khodayari, Nazli

“…Alpha-1 antitrypsin deficiency (AATD) is a genetic disease caused by misfolding and accumulation of mutant alpha-1 antitrypsin (ZAAT) in the endoplasmic…”
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P-C2 T-cell based lentiviral vaccines by Roff, Shannon, Sanou, Missa, Aranyos, Alek, Pu, Ruiyu, Rathore, Mobeen, Levy, Jay, Yamamoto, Janet

“…The failure to elicit high (>80%) protective immunity in recent human and animal trials may be due to the inclusion of vaccine epitopes that enhance lentiviral…”
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P-C2 T-cell based lentiviral vaccines by Roff, Shannon, Sanou, Missa, Aranyos, Alek, Pu, Ruiyu, Rathore, Mobeen, Levy, Jay, Yamamoto, Janet

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Reply by Lu, Yuanqing, Wang, Liqun R., Lee, Jungnam, Mohammad, Naweed S., Aranyos, Alek M., Gould, Calvin, Khodayari, Nazli, Oshins, Regina A., Moneypenny, Craig G., Brantly, Mark L.

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Reply by Lu, Yuanqing, Wang, Liqun R., Lee, Jungnam, Mohammad, Naweed S., Aranyos, Alek M., Gould, Calvin, Khodayari, Nazli, Oshins, Regina A., Moneypenny, Craig G., Brantly, Mark L.

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Reply by Lu, Yuanqing, Wang, Liqun R, Lee, Jungnam, Mohammad, Naweed S, Aranyos, Alek M, Gould, Calvin, Khodayari, Nazli, Oshins, Regina A, Moneypenny, Craig G, Brantly, Mark L

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Reply by Lu, Yuanqing, Wang, Liqun R, Lee, Jungnam, Mohammad, Naweed S, Aranyos, Alek M, Gould, Calvin, Khodayari, Nazli, Oshins, Regina A, Moneypenny, Craig G, Brantly, Mark L

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Reply by Lu, Yuanqing, Wang, Liqun R, Lee, Jungnam, Mohammad, Naweed S, Aranyos, Alek M, Gould, Calvin, Khodayari, Nazli, Oshins, Regina A, Moneypenny, Craig G, Brantly, Mark L

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Reply by Lu, Yuanqing, Wang, Liqun R., Lee, Jungnam, Mohammad, Naweed S., Aranyos, Alek M., Gould, Calvin, Khodayari, Nazli, Oshins, Regina A., Moneypenny, Craig G., Brantly, Mark L.

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Reply by Yuanqing Lu, Liqun R. Wang, Jungnam Lee, Naweed S. Mohammad, Alek M. Aranyos, Calvin Gould, Nazli Khodayari, Regina A. Oshins, Craig G. Moneypenny, Mark L. Brantly

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