Search Results - "Ansari, M.Y."

Parkin clearance of dysfunctional mitochondria regulates ROS levels and increases survival of human chondrocytes by Ansari, M.Y., Khan, N.M., Ahmad, I., Haqqi, T.M.

“…Mitochondrial dysfunction, oxidative stress and chondrocyte death are important contributors to the development and pathogenesis of osteoarthritis (OA). In…”
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Advanced glycation end products (ages) induce stress granule assembly in human OA chondrocytes that captures mRNAS associated with osteoarthritis pathogenesis by Ansari, M.Y, Haqqi, T.M

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Elimination of dysfunctional mitochondria by parkin suppresses oxidative stress and expression of osteoarthritis related genes in human chondrocytes by Ansari, M.Y, Khan, N.M, Ahmad, I, Haqqi, T.M

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Wogonin exerts anti-inflammatory effect by disrupting KEAP-1/NRF2 interactions and activating NRF2 in human OA chondrocytes by Khan, N.M, Ansari, M.Y, Haynie, S, Haqqi, T.M

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A Wogonin-rich Fraction of scutellaria baicalensis Root Extract Exerts Chondroprotective Effects by Suppressing il-1β-induced Activation of AP-1 in Human OA Chondrocytes by Khan, N.M, Haseeb, A, Ansari, M.Y, Haqqi, T.M

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Wogonin exerts anti-inflammatory effect by disrupting keap-1/Nrf2 interactions and activating Nrf2 in human OA chondrocytes by Khan, N.M, Ansari, M.Y, Haynie, S, Haqqi, T.M

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IL-1β induces low levels of autophagy which clears dysfunctional mitochondria and inhibits ROS, MMP-13 and ADAMTS5 expression in OA chondrocytes by Ansari, M.Y, Khan, M.N, Haqqi, T.M

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Autophagy activation by sucrose exhibits chondroprotective effect in human OA chondrocytes by Khan, M.N, Ansari, M.Y, Haqqi, T.M

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Indian medicinal plant butea monosperma flower extract and its bioactive constituent butein activates autophagy in human oa chondrocytes under pathological conditions by Ansari, M.Y, Haynie, S, Haqqi, T.M

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Spinigerin induces apoptotic like cell death in a caspase independent manner in Leishmania donovani by Sardar, A.H., Das, S., Agnihorti, S., Kumar, M., Ghosh, A.K., Abhishek, K., Kumar, A., Purkait, B., Ansari, M.Y., Das, P.

“…•Annexin-V positive and TUNEL positive cells observed upon spinigerin exposure.•ROS machinery was involved in spinigerin mediated apoptosis like…”
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UPREGULATION OF TERMINAL URIDYLYLTRANSFERASE 7 (TUT7) CONTRIBUTES TO IMPAIRED MITOCHONDRIAL DYNAMICS AND DYSFUNCTION IN CHONDROCYTES IN OSTEOARTHRITIS by Ansari, M.Y., Novak, K., Haqqi, T.M.

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GENDER BIAS IN OSTEOARTHRITIS: THE SUSHI CONNECTION by Ansari, M.Y., Novak, K., Haqqi, T.M.

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ERK1/2 mediated activation of dynamin related protein 1 induces mitochondrial network fragmentation, oxidative stress and apoptosis in chondrocytes through cytochrome c release by Ansari, M.Y., Novak, K., Haqqi, T.M.

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Peretinoin protects against IL-1β induced inflammation and matrix degradation in primary human chondrocytes by inhibiting P-38 MAPK signaling by Ahmad, N., Ansari, M.Y., Haqqi, T.M.

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Parkin deficiency impairs the clearance of dysfunctional mitochondria and augments IL-1b induced inflammation in human articular chondrocytes by Ansari, M.Y., Novak, K., Haqqi, T.M.

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ERK1/2-mediated activation of DRP1 regulates mitochondrial dynamics and apoptosis in chondrocytes by Ansari, M.Y., Novak, K., Haqqi, T.M.

“…To determine the Dynamin-related protein 1 (DRP1) regulation of mitochondrial fission in chondrocytes under pathological conditions, an area which is…”
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ZCCHC6 (TUT7) deletion inhibit the expression of IL-6 and MMP-13 and reduces the severity of post-traumatic osteoarthritis in a murine model by Ansari, M.Y., Khan, N.M., Haqqi, T.M.

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Neofunctionalized gene RTl3 and SOX9 collectively modulate COL2A1 expression in human articular chondrocytes by Ball, H.C., Ansari, M.Y., Novak, K., Haqqi, T.M.

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Lysosomal dysfunction in osteoarthritis and aged cartilage induce apoptosis in chondrocytes through bax mediated release of cytochrome C by Ansari, M.Y., Ball, H., Wase, S., Novak, K., Haqqi, T.

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tRNA-derived fragments (tRFs) regulate post-transcriptional gene expression via AGO-dependent mechanism in IL-1β stimulated chondrocytes by Green, J.A., Ansari, M.Y., Ball, H.C., Haqqi, T.M.

“…Recent studies have shown that tRNA-derived RNA fragments (tRFs) are novel regulators of post-transcriptional gene expression. However, the expression profiles…”
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