Search Results - "Amniai, L."

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    Natural Killer Cells Accumulate in Lung-Draining Lymph Nodes and Regulate Airway Eosinophilia in a Murine Model of Asthma by Ple, C, Barrier, M, Amniai, L, Marquillies, P, Bertout, J, Tsicopoulos, A, Walzer, T, Lassalle, P, Duez, C

    Published in Scandinavian journal of immunology (01-08-2010)
    “…Increasing evidence suggests a key role for the innate immune system in asthma development. Although the role of Natural Killer (NK) cells in allergic asthma…”
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    Journal Article
  3. 3

    Recombinant Mycobacterium bovis BCG producing IL‐18 reduces IL‐5 production and bronchoalveolar eosinophilia induced by an allergic reaction by Biet, F., Duez, C., Kremer, L., Marquillies, P., Amniai, L., Tonnel, A.‐B., Locht, C., Pestel, J.

    Published in Allergy (Copenhagen) (01-08-2005)
    “…Background:  Allergic reactions occur through the exacerbated induction of a Th2 cell type expression profile and can be prevented by agents favoring a Th1…”
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    Journal Article
  4. 4

    IL-18 does not increase allergic airway disease in mice when produced by BCG by Amniai, L, Biet, F, Marquillies, P, Locht, C, Pestel, J, Tonnel, A-B, Duez, C

    “…Whilst BCG inhibits allergic airway responses in murine models, IL-18 has adversary effects depending on its environment. We therefore constructed a BCG strain…”
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    Journal Article
  5. 5

    Original article: Recombinant Mycobacterium bovis BCG producing IL-18 reduces IL-5 production and bronchoalveolar eosinophilia induced by an allergic reaction by Biet, F, Duez, C, Kremer, L, Marquillies, P, Amniai, L, A.-B. Tonnel, Locht, C, Pestel, J

    Published in Allergy (Copenhagen) (01-08-2005)
    “…Allergic reactions occur through the exacerbated induction of a Th2 cell type expression profile and can be prevented by agents favoring a Th1 profile…”
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    Journal Article
  6. 6

    Cellules dendritiques et Toll-like récepteurs dans les maladies allergiques by Tonnel, A.B., Rataczak, C., Amniai, L.

    “…La réponse de l'hôte vis-à-vis d'un agent d'agression extérieur (agents infectieux ou allergènes) met en jeu un double système : l'immunité innée et l'immunité…”
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