Search Results - "Ambros, P.F"

234 Genomic analyses of disseminated neuroblastoma cells to study tumor evolution and prognostic markers by Abbasi, M.R, Rifatbegovic, F, Brunner, C, Ladenstein, R, Ambros, I.M, Ambros, P.F

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235 Genomic analyses of circulating tumor DNA in the plasma of neuroblastoma patients by Huetter, S.C, Walder, D, Brunner, C, Ladenstein, R, Abbasi, M.R, Ambros, I.M, Ambros, P.F

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181 INVITED International consensus for neuroblastoma molecular diagnostics: report from the international neuroblastoma risk grouping (INRG) biology committee by Ambros, P.F, Ambros, I.M, Brodeur, G.M, Haber, M, Khan, J, Nakagawara, A, Schleiermacher, G, Speleman, F, Spitz, R, Maris, J.M

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Comparative genetic study of intratumoral heterogenous MYCN amplified neuroblastoma versus aggressive genetic profile neuroblastic tumors by Berbegall, A P, Villamón, E, Piqueras, M, Tadeo, I, Djos, A, Ambros, P F, Martinsson, T, Ambros, I M, Cañete, A, Castel, V, Navarro, S, Noguera, R

“…Intratumoral heterogeneous MYCN amplification (hetMNA) is an unusual event in neuroblastoma with unascertained biological and clinical implications. Diagnosis…”
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Influence of segmental chromosome abnormalities on survival in children over the age of 12 months with unresectable localised peripheral neuroblastic tumours without MYCN amplification by Defferrari, R, Mazzocco, K, Ambros, I M, Ambros, P F, Bedwell, C, Beiske, K, Bénard, J, Berbegall, A P, Bown, N, Combaret, V, Couturier, J, Erminio, G, Gambini, C, Garaventa, A, Gross, N, Haupt, R, Kohler, J, Jeison, M, Lunec, J, Marques, B, Martinsson, T, Noguera, R, Parodi, S, Schleiermacher, G, Tweddle, D A, Valent, A, Van Roy, N, Vicha, A, Villamon, E, Tonini, G P

“…Background: The prognostic impact of segmental chromosome alterations (SCAs) in children older than 1 year, diagnosed with localised unresectable neuroblastoma…”
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Quality Assessment of Genetic Markers Used for Therapy Stratification by Ambros, I.M., Benard, J., Boavida, M., Bown, N., Caron, H., Combaret, V., Couturier, J., Darnfors, C., Delattre, O., Freeman-Edward, J., Gambini, C., Gross, N., Hattinger, C.M., Luegmayr, A., Lunec, J., Martinsson, T., Mazzocco, K., Navarro, S., Noguera, R., O’Neill, S., Pötschger, U., Rumpler, S., Speleman, F., Tonini, G.P., Valent, A., Van Roy, N., Amann, G., De Bernardi, B., Kogner, P., Ladenstein, R., Michon, J., Pearson, A.D.J., Ambros, P.F.

“…Therapy stratification based on genetic markers is becoming increasingly important, which makes commitment to the highest possible reliability of the involved…”
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Sequence based high resolution chromosomal CGH by Kowalska, A, Brunner, B, Bozsaky, E, Chen, Q-R, Stock, C, Lörch, T, Khan, J, Ambros, P F

“…We aimed to directly align a chromosomal CGH (cCGH) pattern with the gene mapping data by taking advantage of the clustering of the GGCC motif at certain…”
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Prognostic impact of deletions at 1p36 and numerical aberrations in Ewing tumors by Hattinger, C.M., Rumpler, S., Strehl, S., Ambros, I.M., Zoubek, A., Pötschger, U., Gadner, H., Ambros, P.F.

“…Ewing's sarcoma, peripheral primitive neuroectodermal tumors, and Askin tumors are referred to as Ewing tumors (ETs), and are characterized by high MIC2…”
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Intratumoural heterogeneity of 1p deletions andMYCN amplification in neuroblastomas by Ambros, P.F., Ambros, I.M., Kerbl, R., Luegmayr, A., Rumpler, S., Ladenstein, R., Amann, G., Kovar, H., Horcher, E., De Bernardi, B., Michon, J., Gadner, H.

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Chromosomal regions involved in the pathogenesis of osteosarcomas by Stock, C., Karger, L., Gadner, H., Ambros, P.F.

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Neuroblastoma cells can actively eliminate supernumerary MYCN gene copies by micronucleus formation—sign of tumour cell revertance? by Ambros, I.M, Rumpler, S, Luegmayr, A, Hattinger, C.M, Strehl, S, Kovar, H, Gadner, H, Ambros, P.F

“…Human neuroblastoma cell lines frequently exhibit MYCN amplification and many are characterised by the presence of morphologically distinct cell types. The…”
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Regression and progression in neuroblastoma. Does genetics predict tumour behaviour? by Ambros, P.F., Ambros, I.M., Strehl, S., Bauer, S., Luegmayr, A., Kovar, H., Ladenstein, R., Fink, F.-M., Horcher, E., Printz, G., Mutz, I., Schilling, F., Urban, C., Gadner, H.

“…Neuroblastoma (NB) is a heterogeneous disease. The clinical course may range from spontaneous regression and maturation to very aggressive behaviour. Stage 4s…”
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Monitoring and functional characterization of rare tumor cells in the haematopoietic system by Mehes, G., Hattinger, C.M., Lörch, T., Luegmayr, A., Gadner, H., Ambros, P.F.

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Tumour cells can eliminate amplified genes by Ambros, A.I., Luegmayr, A., Rumpler, S., Hattinger, C.M., Kovar, H., Gadner, H., Ambros, P.F.

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Disseminated choriocarcinoma in infancy is curable by chemotherapy and delayed tumour resection by Blohm, M.E.G, Calaminus, G, Gnekow, A.K, Heidemann, P.H, Bolkenius, M, Weinel, P, von Schweinitz, D, Ambros, P.F, Schneider, D.T, Harms, D, Göbel, U

“…Infantile choriocarcinoma has a poor prognosis with only 2 surviving children reported in the literature. 2 additional successfully treated children are…”
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Prognostic implications of tumor cell infiltration of the hematopoietic system in pediatric and adult tumor patients by Ambros, P.F., Mehes, G., Luegmayr, A., Witt, A., Kubista, E., Hattinger, C., Lörch, T., Ambros, I.M., Gadner, H.

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Fluorescence in situ hybridization combined with immunohistochemistry for highly sensitive detection of chromosome 1 aberrations in neuroblastoma by Strehl, S, Ambros, P F

“…The development and application of a highly sensitive double-target fluorescence in situ hybridization (FISH) method in combination with immunohistochemistry…”
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Schwann cells in neuroblastoma by Ambros, I.M., Ambros, P.F.

“…Why should we consider Schwann cells when we are interested in the biology of neuroblastomas (NBs)? Although we are familiar with the term “stroma-rich” NB, we…”
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Fine-mapping of cytogenetically undetectable EWS/ERG fusions on DNA fibers of Ewing tumors by Hattinger, C M, Rumpler, S, Kovar, H, Ambros, P F

“…In contrast to the EWS/FLI1 fusion which is represented by a t(11;22)(q24;q12), EWS/ERG fusions are frequently cytogenetically not detectable. Three Ewing…”
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Homology-dependent gene silencing in transgenic plants: epistatic silencing loci contain multiple copies of methylated transgenes by Matzke, A J, Neuhuber, F, Park, Y D, Ambros, P F, Matzke, M A

“…Previous work has shown that two homologous, unlinked transgene loci can interact in plant nuclei, leading to non-reciprocal trans-inactivation and methylation…”
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