Search Results - "Ahn, Bong W"
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EGCG Blocks Tumor Promoter-induced MMP-9 Expression Via Suppression of MAPK and AP-1 Activation in Human Gastric AGS Cells
Published in Anticancer research (01-03-2004)“…Overexpression of matrix metalloproteinases (MMPs) has been known to correlate closely with tumor cell invasion and strategies to down-regulate their…”
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Regulation of urokinase plasminogen activator by epigallocatechin-3-gallate in human fibrosarcoma cells
Published in European journal of pharmacology (08-03-2004)“…(−)-Epigallocatechin-3-gallate (EGCG), a main flavanol of green tea, potently suppressed the urokinase-type plasminogen activator (uPA) expression in human…”
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Interleukin-1β stimulates IL-8 expression through MAP kinase and ROS signaling in human gastric carcinoma cells
Published in Oncogene (26-08-2004)“…Recent studies have suggested that the expression of interleukin-8 (IL-8) directly correlates with the vascularity of human gastric carcinomas. In this study,…”
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Triptolide inhibits tumor promoter-induced uPAR expression via blocking NF-κB signaling in human gastric AGS cells
Published in Anticancer research (01-09-2007)Get full text
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IL-1β induces MMP-9 via reactive oxygen species and NF-κB in murine macrophage RAW 264.7 cells
Published in Biochemical and biophysical research communications (25-10-2002)“…IL-1β increased the production of proenzyme of MMP-9 (pro-MMP-9) in a time- and dose-dependent manner in murine macrophage RAW 264.7 cells. However, the…”
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Involvement of NF-kappaB and caspases in silibinin-induced apoptosis of endothelial cells
Published in International journal of molecular medicine (01-01-2004)“…Silibinin, the flavonoid found in the milk thistle, has been shown to suppress cell growth and exhibit anti-cancer effects. Some flavonoids were reported to…”
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Epigallocatechin-3-gallate inhibits the PDGF-induced VEGF expression in human vascular smooth muscle cells via blocking PDGF receptor and Erk-1/2
Published in International journal of oncology (01-11-2006)“…Platelet-derived growth factor (PDGF) has been known to induce vascular endothelial growth factor (VEGF) expression in human vascular smooth muscle cells…”
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Extracellular signal-regulated kinase and AP-1 pathways are involved in reactive oxygen species-induced urokinase plasminogen activator receptor expression in human gastric cancer cells
Published in International journal of oncology (01-06-2005)“…Overexpression of urokinase plasminogen activator receptor (uPAR) is known to correlate closely with tumor cell invasion and metastasis. In gastric cancer,…”
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Extracellular signal-regulated kinases and AP-1 mediate the up-regulation of vascular endothelial growth factor by PDGF in human vascular smooth muscle cells
Published in International journal of oncology (2006)“…Endothelial cells (ECs) and vascular smooth muscle cells (VSMCs) have been shown to communicate with each other via cytokine signaling during…”
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Urokinase plasminogen activator receptor is upregulated by Helicobacter pylori in human gastric cancer AGS cells via ERK, JNK, and AP-1
Published in Biochemical and biophysical research communications (05-08-2005)“…The gastric pathogen Helicobacter pylori ( H. pylori) is suggested to be associated with gastric cancer progression. In this study, we investigated the effect…”
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Induction of apoptosis by the green tea flavonol (-)-epigallocatechin-3-gallate in human endothelial ECV 304 cells
Published in Anticancer research (01-11-2002)“…We have previously shown that treatment with (-)-epigallocatechin-3-gallate (EGCG) inhibited vascularity and tumor growth in human colon cancer xenografts in…”
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P38 MAPK pathway is involved in the urokinase plasminogen activator expression in human gastric SNU-638 cells
Published in Oncology reports (01-09-2003)“…Overexpression of urokinase plasminogen activator (uPA) is known to correlate closely with tumor cell invasion and metastasis. In gastric cancer, however, the…”
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(−)-Epigallocatechin-3-gallate inhibits monocyte chemotactic protein-1 expression in endothelial cells via blocking NF-κB signaling
Published in Life sciences (1973) (01-05-2007)“…Monocyte chemotactic protein-1 (MCP-1) is a potent chemoattractant for monocytes and plays a key role in various inflammatory responses, including…”
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EGF stimulates uPAR expression and cell invasiveness through ERK, AP-1, and NF-κB signaling in human gastric carcinoma cells
Published in Oncology reports (01-12-2008)Get full text
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Helicobacter pylori stimulates urokinase plasminogen activator receptor expression and cell invasiveness through reactive oxygen species and NF-κB signaling in human gastric carcinoma cells
Published in International journal of molecular medicine (01-04-2007)“…The gastric pathogen, helicobacter pylori (H. pylori), has been associated with the progression of gastric cancer. It was previously reported that H. pylori…”
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EGF stimulates uPAR expression and cell invasiveness through ERK, AP-1, and NF-kappaB signaling in human gastric carcinoma cells
Published in Oncology reports (01-12-2008)“…Overexpression of epidermal growth factor (EGF) and urokinase plasminogen activator receptor (uPAR) have been observed in human gastric cancers. However, the…”
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(-)-Epigallocatechin-3-gallate inhibits monocyte chemotactic protein-1 expression in endothelial cells via blocking NF-kappaB signaling
Published in Life sciences (1973) (01-05-2007)“…Monocyte chemotactic protein-1 (MCP-1) is a potent chemoattractant for monocytes and plays a key role in various inflammatory responses, including…”
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18
Triptolide inhibits tumor promoter-induced uPAR expression via blocking NF-kappaB signaling in human gastric AGS cells
Published in Anticancer research (01-09-2007)“…The overexpression of urokinase-type plasminogen activator receptor (uPAR) is closely related to tumor cell invasion. Therefore, strategies for down-regulating…”
Get full text
Journal Article -
19
Triptolide Inhibits Tumor Promoter-induced uPAR Expression via Blocking NF-κB Signaling in Human Gastric AGS Cells
Published in Anticancer research (01-09-2007)“…The overexpression of urokinase-type plasminogen activator receptor (uPAR) is closely related to tumor cell invasion. Therefore, strategies for down-regulating…”
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