Search Results - "Abrial, M"

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    The SR/ER-mitochondria calcium crosstalk is regulated by GSK3β during reperfusion injury by Gomez, L, Thiebaut, P-A, Paillard, M, Ducreux, S, Abrial, M, Crola Da Silva, C, Durand, A, Alam, M R, Van Coppenolle, F, Sheu, S-S, Ovize, M

    Published in Cell death and differentiation (01-02-2016)
    “…Glycogen synthase kinase-3 β (GSK3 β ) is a multifunctional kinase whose inhibition is known to limit myocardial ischemia–reperfusion injury. However, the…”
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    Journal Article
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    Therapeutic Hypothermia After Cardiac Arrest: Involvement of the Risk Pathway in Mitochondrial PTP-Mediated Neuroprotection by Jahandiez, Vincent, Cour, Martin, Abrial, Maryline, Loufouat, Joseph, Ovize, Michel, Argaud, Laurent

    Published in Shock (Augusta, Ga.) (01-08-2019)
    “…ABSTRACTTherapeutic hypothermia is neuroprotective after cardiac arrest (CA) via poorly understood mechanisms. It may prevent mitochondrial permeability…”
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    Journal Article
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    Cardiac fibroblasts protect cardiomyocytes against lethal ischemia–reperfusion injury by Abrial, Maryline, Da Silva, Claire Crola, Pillot, Bruno, Augeul, Lionel, Ivanes, Fabrice, Teixeira, Geoffrey, Cartier, Régine, Angoulvant, Denis, Ovize, Michel, Ferrera, René

    “…Abstract Roles of cardiac fibroblasts (CFs) in the regulation of myocardial structure and function have been emphasized in the last decade. Their implications…”
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    Journal Article
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    Ubiquitous protective effects of cyclosporine A in preventing cardiac arrest-induced multiple organ failure by Cour, Martin, Abrial, Maryline, Jahandiez, Vincent, Loufouat, Joseph, Belaïdi, Elise, Gharib, Abdallah, Varennes, Annie, Monneret, Guillaume, Thibault, Hélène, Ovize, Michel, Argaud, Laurent

    Published in Journal of applied physiology (1985) (15-10-2014)
    “…Opening of the mitochondrial permeability transition pore (mPTP) appears to be a pivotal event in myocardial ischemia-reperfusion (I/R) injury. Resuscitated…”
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    Journal Article
  7. 7

    The SR/ER-mitochondria calcium crosstalk is regulated by GSK3[beta] during reperfusion injury by Gomez, L, Thiebaut, P-a, Paillard, M, Ducreux, S, Abrial, M, Crola Da Silva, C, Durand, A, Alam, M R, Van Coppenolle, F, Sheu, S-s, Ovize, M

    Published in Cell death and differentiation (01-02-2016)
    “…Glycogen synthase kinase-3β (GSK3β) is a multifunctional kinase whose inhibition is known to limit myocardial ischemia-reperfusion injury. However, the…”
    Get full text
    Journal Article
  8. 8

    The SR/ER-mitochondria calcium crosstalk is regulated by GSK3beta during reperfusion injury by Gomez, L., Thiebaut, P. A., Paillard, M., Ducreux, S., Abrial, M., Crola da Silva, C., Durand, A., Alam, M. R., van Coppenolle, F., Sheu, S. S., Ovize, Michel

    Published in Cell death and differentiation (01-02-2016)
    “…Glycogen synthase kinase-3beta (GSK3beta) is a multifunctional kinase whose inhibition is known to limit myocardial ischemia-reperfusion injury. However, the…”
    Get full text
    Journal Article
  9. 9

    The basis of ischemia-reperfusion and myocardial protection by Benhabbouche, S, Crola da Silva, C, Abrial, M, Ferrera, R

    “…The myocardial infarction represents a major cause of mortality. The deleterious phenomena arising during the ischaemia and the reperfusion of the myocardium…”
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    Journal Article
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    Erratum: The SR/ER-mitochondria calcium crosstalk is regulated by GSK3β during reperfusion injury by Gomez, L, Thiebaut, P-A, Paillard, M, Ducreux, S, Abrial, M, Crola Da Silva, C, Durand, A, Alam, M R, Van Coppenolle, F, Sheu, S-S, Ovize, M

    Published in Cell death and differentiation (05-10-2015)
    “…Correction to: Cell Death and Differentiation advance online publication 24 July 2015; doi: 10.1038/cdd.2015.101 In this article, The SR/ER-mitochondria…”
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    Journal Article
  13. 13

    Base des phénomènes d’ischémie reperfusion et de la protection myocardique by Benhabbouche, S., Crola da silva, C., Abrial, M., Ferrera, R.

    “…L’infarctus du myocarde constitue une cause majeure de mortalité. Les phénomènes délétères survenant pendant l’ischémie et la reperfusion du myocarde sont…”
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    Journal Article Conference Proceeding
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